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  1. Reduced DPP4 Binding Confers Resistance to Soluble DPP4 While Preserving MERS-CoV Entry into Cells Expressing High Levels of DPP4
  2. Update on BA.3.2 epidemiology: Evidence for co-circulation and preferential infection of children
  3. The conserved QTQTX motif in the SARS-CoV-2 spike protein is dispensable for cleavage and lung cell entry of the emerging variant BA.3.2
  4. JN.1-adapted vaccination is associated with readjustment of ancestral memory B cells toward neutralization within the JN.1 antigenic space
  5. The conserved QTQTX motif in the SARS-CoV-2 spike protein is dispensable for cleavage and lung cell entry of the emerging variant BA.3.2
  6. Lock out: targeting TMPRSS2 to block influenza and coronaviruses
  7. A mutation in the spike protein of MERS-CoV confers resistance against soluble DPP4
  8. Development of immortalized Callithrix jacchus kidney cell lines supporting infection with a panel of viruses
  9. Development of immortalized Callithrix jacchus kidney cell lines supporting infection with a panel of viruses
  10. Amino acid residues 655 and 969 in the spike protein of Omicron subvariant BA.1 control use of TMPRSS2 versus Cathepsin L dependent entry pathways and cell tropism
  11. Polyvalent Mannuronic Acid-Coated Gold Nanoparticles for Probing Multivalent Lectin–Glycan Interaction and Blocking Virus Infection
  12. Simulations and active learning enable efficient identification of an experimentally-validated broad coronavirus inhibitor
  13. Q1020R in the spike proteins of MERS-CoV from Arabian camels and recent human cases confers resistance against soluble human DPP4
  14. Entry Efficiency, Protease Dependence, and Antibody-Mediated Neutralization of SARS-CoV-2 Sublineages KP.3.1.1 and XEC
  15. Immune Response to SARS-CoV-2 XBB.1.5 and JN.1 Variants Following XBB.1.5 Booster Vaccination in Liver Transplant Recipients
  16. Comparative Analysis of Host Cell Entry Efficiency and Neutralization Sensitivity of Emerging SARS-CoV-2 Lineages KP.2, KP.2.3, KP.3, and LB.1
  17. Impact of JN.1 booster vaccination on neutralisation of SARS-CoV-2 variants KP.3.1.1 and XEC
  18. Interferon-Stimulated Genes that Target Retrovirus Translation
  19. Development of rhesus macaque astrocyte cell lines supporting infection with a panel of viruses
  20. Virological Traits of the SARS-CoV-2 BA.2.87.1 Lineage
  21. The Inhibition of Gag-Pol Expression by the Restriction Factor Shiftless Is Dispensable for the Restriction of HIV-1 Infection
  22. Diversification of the VH3‐53 immunoglobulin gene segment by somatic hypermutation results in neutralization of SARS‐CoV‐2 virus variants
  23. Immune responses following BNT162b2 XBB.1.5 vaccination in patients on haemodialysis in Germany
  24. SARS-CoV-2 BA.2.86 enters lung cells and evades neutralizing antibodies with high efficiency
  25. Host cell entry and neutralisation sensitivity of the SARS-CoV-2 XBB.1.16 lineage
  26. Development of immortalized rhesus macaque kidney cells supporting infection with a panel of viruses
  27. Discovery of Polyphenolic Natural Products as SARS-CoV-2 Mpro Inhibitors for COVID-19
  28. TMPRSS2 Is Essential for SARS-CoV-2 Beta and Omicron Infection
  29. Primate Simplexviruses Differ in Tropism for Macaque Cells
  30. Discovery of Polyphenolic Natural Products as SARS-CoV-2 Mpro Inhibitors for COVID-19
  31. The SARS-CoV-2 Delta-Omicron Recombinant Lineage (XD) Exhibits Immune-Escape Properties Similar to the Omicron (BA.1) Variant
  32. Host Cell Entry and Neutralization Sensitivity of SARS-CoV-2 Lineages B.1.620 and R.1
  33. Combinations of Host- and Virus-Targeting Antiviral Drugs Confer Synergistic Suppression of SARS-CoV-2
  34. Publisher Correction: Dalbavancin: novel candidate for COVID-19 treatment
  35. BNT162b2-boosted immune responses six months after heterologous or homologous ChAdOx1nCoV-19/BNT162b2 vaccination against COVID-19
  36. BNT162b2 booster after heterologous prime-boost vaccination induces potent neutralizing antibodies and T cell reactivity against SARS-CoV-2 Omicron BA.1 in young adults
  37. Nafamostat-Mediated Inhibition of SARS-CoV-2 Ribosomal Frameshifting Is Insufficient to Impair Viral Replication in Vero Cells. Comment on Munshi et al. Identifying Inhibitors of −1 Programmed Ribosomal Frameshifting in a Broad Spectrum of Coronaviruse...
  38. Peptidomimetic inhibitors of TMPRSS2 block SARS-CoV-2 infection in cell culture
  39. Mutagenic Analysis of the HIV Restriction Factor Shiftless
  40. Evidence for an ACE2-Independent Entry Pathway That Can Protect from Neutralization by an Antibody Used for COVID-19 Therapy
  41. Small-Molecule Thioesters as SARS-CoV-2 Main Protease Inhibitors: Enzyme Inhibition, Structure–Activity Relationships, Antiviral Activity, and X-ray Structure Determination
  42. Comparable neutralisation evasion of SARS-CoV-2 omicron subvariants BA.1, BA.2, and BA.3
  43. Efficient antibody evasion but reduced ACE2 binding by the emerging SARS-CoV-2 variant B.1.640.2
  44. Understanding Omicron: Transmissibility, immune evasion and antiviral intervention
  45. SARS-CoV-2 Omicron sublineages show comparable cell entry but differential neutralization by therapeutic antibodies
  46. SARS-CoV-2 variants C.1.2 and B.1.621 (Mu) partially evade neutralization by antibodies elicited upon infection or vaccination
  47. Investigations on SARS-CoV-2 Susceptibility of Domestic and Wild Animals Using Primary Cell Culture Models Derived from the Upper and Lower Respiratory Tract
  48. Omicron: Master of immune evasion maintains robust ACE2 binding
  49. Functional analysis of polymorphisms at the S1/S2 site of SARS-CoV-2 spike protein
  50. Augmented neutralization of SARS‐CoV‐2 Omicron variant by boost vaccination and monoclonal antibodies
  51. Neutralizing antibody responses 300 days after SARS‐CoV‐2 infection and induction of high antibody titers after vaccination
  52. Alternatives to animal models and their application in the discovery of species susceptibility to SARS-CoV-2 and other respiratory infectious pathogens: A review
  53. The Omicron variant is highly resistant against antibody-mediated neutralization: Implications for control of the COVID-19 pandemic
  54. Rapid SARS-CoV-2 Adaptation to Available Cellular Proteases
  55. MCMV-based vaccine vectors expressing full-length viral proteins provide long-term humoral immune protection upon a single-shot vaccination
  56. No evidence for increased cell entry or antibody evasion by Delta sublineage AY.4.2
  57. A Recombinant System and Reporter Viruses for Papiine Alphaherpesvirus 2
  58. The MEK1/2-inhibitor ATR-002 efficiently blocks SARS-CoV-2 propagation and alleviates pro-inflammatory cytokine/chemokine responses
  59. Heterologous ChAdOx1 nCoV-19 and BNT162b2 prime-boost vaccination elicits potent neutralizing antibody responses and T cell reactivity against prevalent SARS-CoV-2 variants
  60. Activation of Sphingomyelinase-Ceramide-Pathway in COVID-19 Purposes Its Inhibition for Therapeutic Strategies
  61. Novel SARS-CoV-2 receptors: ASGR1 and KREMEN1
  62. Erythrocytes increase endogenous sphingosine 1-phosphate levels as an adaptive response to SARS-CoV-2 infection
  63. Improved cellular and humoral immunity upon a second BNT162b2 and mRNA-1273 boost in prime-boost vaccination no/low responders with end-stage renal disease
  64. Protective mucosal immunity against SARS-CoV-2 after heterologous systemic prime-mucosal boost immunization
  65. Spike residue 403 affects binding of coronavirus spikes to human ACE2
  66. The spike protein of SARS-CoV-2 variant A.30 is heavily mutated and evades vaccine-induced antibodies with high efficiency
  67. Evidence that two instead of one defective interfering RNA in influenza A virus-derived defective interfering particles (DIPs) does not enhance antiviral activity
  68. A novel class of TMPRSS2 inhibitors potently block SARS-CoV-2 and MERS-CoV viral entry and protect human epithelial lung cells
  69. Delta variant (B.1.617.2) sublineages do not show increased neutralization resistance
  70. B.1.617.2 enters and fuses lung cells with increased efficiency and evades antibodies induced by infection and vaccination
  71. The Upper Respiratory Tract of Felids Is Highly Susceptible to SARS-CoV-2 Infection
  72. A pair of noncompeting neutralizing human monoclonal antibodies protecting from disease in a SARS‐CoV‐2 infection model
  73. Functional comparison of MERS-coronavirus lineages reveals increased replicative fitness of the recombinant lineage 5
  74. SARS-CoV-2 delta variant neutralisation after heterologous ChAdOx1-S/BNT162b2 vaccination
  75. Neutralization of the SARS-CoV-2 Delta variant after heterologous and homologous BNT162b2 or ChAdOx1 nCoV-19 vaccination
  76. Immune responses against SARS-CoV-2 variants after heterologous and homologous ChAdOx1 nCoV-19/BNT162b2 vaccination
  77. SARS-CoV-2 neutralizing antibodies: Longevity, breadth, and evasion by emerging viral variants
  78. SARS-CoV-2 variant B.1.617 is resistant to bamlanivimab and evades antibodies induced by infection and vaccination
  79. How SARS-CoV-2 makes the cut
  80. Humoral and Cellular Immune Responses Against Severe Acute Respiratory Syndrome Coronavirus 2 Variants and Human Coronaviruses After Single BNT162b2 Vaccination
  81. Urinary Levels of SARS-CoV-2 Nucleocapsid Protein Associate With Risk of AKI and COVID-19 Severity: A Single-Center Observational Study
  82. Therapeutic Application of alpha-1-antitrypsin in COVID-19
  83. Cell culture-based production and in vivo characterization of purely clonal defective interfering influenza virus particles
  84. The SARS-CoV-2 and other human coronavirus spike proteins are fine-tuned towards temperature and proteases of the human airways
  85. SARS-CoV-2 mutations acquired in mink reduce antibody-mediated neutralization
  86. The sphingosine kinase 1 activator, K6PC-5, attenuates Ebola virus infection
  87. SARS-CoV-2 variants B.1.351 and P.1 escape from neutralizing antibodies
  88. Alpha-1 antitrypsin inhibits TMPRSS2 protease activity and SARS-CoV-2 infection
  89. Ex vivo assay to evaluate the efficacy of drugs targeting sphingolipids in preventing SARS-CoV-2 infection of nasal epithelial cells
  90. Mutation D614G increases SARS-CoV-2 transmission
  91. Camostat mesylate inhibits SARS-CoV-2 activation by TMPRSS2-related proteases and its metabolite GBPA exerts antiviral activity
  92. Dalbavancin: novel candidate for COVID-19 treatment
  93. Natural cystatin C fragments inhibit GPR15-mediated HIV and SIV infection without interfering with GPR15L signaling
  94. Molecular mechanism of inhibiting the SARS-CoV-2 cell entry facilitator TMPRSS2 with camostat and nafamostat
  95. Inhibition of acid sphingomyelinase by ambroxol prevents SARS-CoV-2 entry into epithelial cells
  96. Synergistic inhibition of SARS-CoV-2 cell entry by otamixaban and covalent protease inhibitors: pre-clinical assessment of pharmacological and molecular properties
  97. The SARS-CoV-2 and other human coronavirus spike proteins are fine-tuned towards temperature and proteases of the human airways
  98. Low serum neutralizing anti-SARS-CoV-2 S antibody levels in mildly affected COVID-19 convalescent patients revealed by two different detection methods
  99. Corrigendum: Compact, Polyvalent Mannose Quantum Dots as Sensitive, Ratiometric FRET Probes for Multivalent Protein–Ligand Interactions
  100. Camostat Mesylate May Reduce Severity of Coronavirus Disease 2019 Sepsis: A First Observation
  101. Pharmacological Inhibition of Acid Sphingomyelinase Prevents Uptake of SARS-CoV-2 by Epithelial Cells
  102. Sphingosine prevents binding of SARS–CoV-2 spike to its cellular receptor ACE2
  103. Glycan-Gold Nanoparticles as Multifunctional Probes for Multivalent Lectin–Carbohydrate Binding: Implications for Blocking Virus Infection and Nanoparticle Assembly
  104. LY6E impairs coronavirus fusion and confers immune control of viral disease
  105. Chloroquine does not inhibit SARS-CoV-2
  106. Probing Multivalent Lectin-Carbohydrate Binding via Multifunctional Glycan-Gold Nanoparticles: Implications for Blocking Virus Infection
  107. Probing Multivalent Lectin-Carbohydrate Binding via Multifunctional Glycan-Gold Nanoparticles: Implications for Blocking Virus Infection
  108. Furin cleavage of SARS-CoV-2 spike is required for infection of lung cells
  109. Structural Basis for Potent Neutralization of Betacoronaviruses by Single-Domain Camelid Antibodies
  110. H2 influenza A virus is not pathogenic in Tmprss2 knock-out mice
  111. Nafamostat inhibits SARS-CoV-2
  112. SARS-CoV-2 uses ACE2 and TMPRSS2 for infection of lung cells
  113. SARS-CoV-2 uses ACE2 and TMPRSS2 for infection of lung cells
  114. Polymorphisms in dipeptidyl peptidase 4 reduce host cell entry of Middle East respiratory syndrome coronavirus
  115. A Fosmid-Based System for the Generation of Recombinant Cercopithecine Alphaherpesvirus 2 Encoding Reporter Genes
  116. Role of rhesus macaque IFITM3(2) in simian immunodeficiency virus infection of macaques
  117. Analysis of Resistance of Ebola Virus Glycoprotein-Driven Entry Against MDL28170, An Inhibitor of Cysteine Cathepsins
  118. Hemagglutinin Cleavability, Acid Stability, and Temperature Dependence Optimize Influenza B Virus for Replication in Human Airways
  119. Spike proteins of novel MERS-coronavirus isolates from North- and West-African dromedary camels mediate robust viral entry into human target cells
  120. Evidence for influenza B virus hemagglutinin adaptation to the human host: high cleavability, acid-stability and preference for cool temperature
  121. Analysis of IFITM-IFITM Interactions by a Flow Cytometry-Based FRET Assay
  122. Kaposi sarcoma in a guereza monkey
  123. Novel Virus Related to Kaposi’s Sarcoma–Associated Herpesvirus from Colobus Monkey
  124. Evidence that Calu-3 cells are largely resistant to Ebola virus entry
  125. Tmprss2 knock-out mice are resistant to H10 influenza A virus pathogenesis
  126. IFITM s mediate viral evasion in acute and chronic hepatitis C virus infection
  127. Guanylate-Binding Proteins 2 and 5 Exert Broad Antiviral Activity by Inhibiting Furin-Mediated Processing of Viral Envelope Proteins
  128. SPP and SKI-1 inhibitor block Ebola virus entry
  129. Modulation of HIV-1 Gag/Gag-Pol frameshifting by tRNA abundance
  130. Detection of antibodies against viruses in a macaque colony using a chip-based approach
  131. The cell Line SH-SY5Y is resistant to infection by Ebola virus and other filoviruses
  132. A safe system for production of defective interfering particles
  133. Virosomes interfere with Ebola virus control by the immune system
  134. Focal epithelial hyperplasia and papillomavirus infection in a bonobo
  135. Development and use of Lentiviral Vectors Pseudotyped with Influenza B Haemagglutinins: application to vaccine immunogenicity, mAb potency and sero-surveillance studies
  136. Tetherin efficiently inhibits Nipah virus but not Ebola virus spread in fruit bat cells
  137. MERS-coronavirus spike protein: Importance of protease cleavage sites for viral entry
  138. A MERS-coronavirus variant that is partially resistant against neutralizing antibodies
  139. The cellular protease TMPRSS11A can convert influenza virus into an infectious form
  140. Cell Entry of Influenza A Viruses: Sweet Talk between HA and Ca V 1.2
  141. A GXXXA motif in the Ebola virus glycoprotein is required for tetherin antagonism
  142. Attachment/Binding
  143. Priming Time: How Cellular Proteases Arm Coronavirus Spike Proteins
  144. VSV-G antagonizes tetherin in transfected cells
  145. Inhibition of lectin-dependent enhancement of Ebola virus entry into cells
  146. Identification of sites in SARS-S required cleavage and activation by TMPRSS2
  147. TMPRSS2 of non-human primates activates influenza viruses
  148. Herpes B virus replication and viral lesions in the liver of a cynomolgus macaque which died from severe disease with rapid onset
  149. pH Optimum of Hemagglutinin-Mediated Membrane Fusion Determines Sensitivity of Influenza A Viruses to the Interferon-Induced Antiviral State and IFITMs
  150. Rhesus macaque IFITM3 gene polymorphisms and SIV infection
  151. A Polymorphism within the Internal Fusion Loop of the Ebola Virus Glycoprotein Modulates Host Cell Entry
  152. Detection systems for antibody responses against herpes B virus
  153. Evidence that Processing of the Severe Fever with Thrombocytopenia Syndrome Virus Gn/Gc Polyprotein Is Critical for Viral Infectivity and Requires an Internal Gc Signal Peptide
  154. Virion Background and Efficiency of Virion Incorporation Determine Susceptibility of Simian Immunodeficiency Virus Env-Driven Viral Entry to Inhibition by IFITM Proteins
  155. The Tetherin Antagonism of the Ebola Virus Glycoprotein Requires an Intact Receptor-Binding Domain and Can Be Blocked by GP1-Specific Antibodies
  156. The Role of Phlebovirus Glycoproteins in Viral Entry, Assembly and Release
  157. The Hemagglutinin of Bat-Associated Influenza Viruses Is Activated by TMPRSS2 for pH-Dependent Entry into Bat but Not Human Cells
  158. Compact, Polyvalent Mannose Quantum Dots as Sensitive, Ratiometric FRET Probes for Multivalent Protein-Ligand Interactions
  159. Compact, Polyvalent Mannose Quantum Dots as Sensitive, Ratiometric FRET Probes for Multivalent Protein-Ligand Interactions
  160. The Glycoproteins of All Filovirus Species Use the Same Host Factors for Entry into Bat and Human Cells but Entry Efficiency Is Species Dependent
  161. The Proteolytic Activation of (H3N2) Influenza A Virus Hemagglutinin Is Facilitated by Different Type II Transmembrane Serine Proteases
  162. TMPRSS2 Isoform 1 Activates Respiratory Viruses and Is Expressed in Viral Target Cells
  163. Exclusive Decoration of Simian Immunodeficiency Virus Env with High-Mannose Type N-Glycans Is Not Compatible with Mucosal Transmission in Rhesus Macaques
  164. Tetherin Sensitivity of Influenza A Viruses Is Strain Specific: Role of Hemagglutinin and Neuraminidase
  165. Interferon-Induced Transmembrane Protein–Mediated Inhibition of Host Cell Entry of Ebolaviruses
  166. Analysis of Ebola Virus Entry Into Macrophages
  167. Comparative Analysis of Host Cell Entry of Ebola Virus From Sierra Leone, 2014, and Zaire, 1976
  168. Protease inhibitors targeting coronavirus and filovirus entry
  169. IFITM Proteins Inhibit Entry Driven by the MERS-Coronavirus Spike Protein: Evidence for Cholesterol-Independent Mechanisms
  170. DESC1 and MSPL Activate Influenza A Viruses and Emerging Coronaviruses for Host Cell Entry
  171. Inhibition of Proprotein Convertases Abrogates Processing of the Middle Eastern Respiratory Syndrome Coronavirus Spike Protein in Infected Cells but Does Not Reduce Viral Infectivity
  172. Bitter-sweet symphony: glycan–lectin interactions in virus biology
  173. Influenza A Virus Encoding Secreted Gaussia Luciferase as Useful Tool to Analyze Viral Replication and Its Inhibition by Antiviral Compounds and Cellular Proteins
  174. Analysis of Determinants in Filovirus Glycoproteins Required for Tetherin Antagonism
  175. The clinically approved drugs amiodarone, dronedarone and verapamil inhibit filovirus cell entry
  176. Induced and spontaneous heart rate turbulence in mice: influence of coupling interval
  177. Toll-Like Receptor 3 Signalling Up-Regulates Expression of the HIV Co-Receptor G-Protein Coupled Receptor 15 on Human CD4+ T Cells
  178. Tmprss2 Is Essential for Influenza H1N1 Virus Pathogenesis in Mice
  179. Lack of MERS Coronavirus Neutralizing Antibodies in Humans, Eastern Province, Saudi Arabia
  180. Proteolytic activation of the SARS-coronavirus spike protein: Cutting enzymes at the cutting edge of antiviral research
  181. TMPRSS2 and ADAM17 Cleave ACE2 Differentially and Only Proteolysis by TMPRSS2 Augments Entry Driven by the Severe Acute Respiratory Syndrome Coronavirus Spike Protein
  182. Platelet activation suppresses HIV-1 infection of T cells
  183. TMPRSS2 Activates the Human Coronavirus 229E for Cathepsin-Independent Host Cell Entry and Is Expressed in Viral Target Cells in the Respiratory Epithelium
  184. The Spike Protein of the Emerging Betacoronavirus EMC Uses a Novel Coronavirus Receptor for Entry, Can Be Activated by TMPRSS2, and Is Targeted by Neutralizing Antibodies
  185. Severe Fever with Thrombocytopenia Virus Glycoproteins Are Targeted by Neutralizing Antibodies and Can Use DC-SIGN as a Receptor for pH-Dependent Entry into Human and Animal Cell Lines
  186. Cellular Entry of Retroviruses
  187. Attachment/Binding
  188. Host Cell Factors in Filovirus Entry: Novel Players, New Insights
  189. The role of the alternative coreceptor GPR15 in SIV tropism for human cells
  190. How Ebola Virus Counters the Interferon System
  191. Influenza A Virus Does Not Encode a Tetherin Antagonist with Vpu-Like Activity and Induces IFN-Dependent Tetherin Expression in Infected Cells
  192. Influenza and SARS-Coronavirus Activating Proteases TMPRSS2 and HAT Are Expressed at Multiple Sites in Human Respiratory and Gastrointestinal Tracts
  193. Cathepsins B and L activate Ebola but not Marburg virus glycoproteins for efficient entry into cell lines and macrophages independent of TMPRSS2 expression
  194. CD4- and dynamin-dependent endocytosis of HIV-1 into plasmacytoid dendritic cells
  195. Comparative Analysis of Ebola Virus Glycoprotein Interactions With Human and Bat Cells
  196. The Ebola Virus Glycoprotein and HIV-1 Vpu Employ Different Strategies to Counteract the Antiviral Factor Tetherin
  197. The SARS-Coronavirus-Host Interactome: Identification of Cyclophilins as Target for Pan-Coronavirus Inhibitors
  198. Cleavage and Activation of the Severe Acute Respiratory Syndrome Coronavirus Spike Protein by Human Airway Trypsin-Like Protease
  199. DC-SIGN: Access Portal for Sweet Viral Killers
  200. Different host cell proteases activate the SARS-coronavirus spike-protein for cell–cell and virus–cell fusion
  201. Evidence that TMPRSS2 Activates the Severe Acute Respiratory Syndrome Coronavirus Spike Protein for Membrane Fusion and Reduces Viral Control by the Humoral Immune Response
  202. Mouse LSECtin as a model for a human Ebola virus receptor
  203. The multiple facets of HIV attachment to dendritic cell lectins
  204. Novel insights into proteolytic cleavage of influenza virus hemagglutinin
  205. TMPRSS2 and TMPRSS4 Facilitate Trypsin-Independent Spread of Influenza Virus in Caco-2 Cells
  206. A Single Asparagine-Linked Glycosylation Site of the Severe Acute Respiratory Syndrome Coronavirus Spike Glycoprotein Facilitates Inhibition by Mannose-Binding Lectin through Multiple Mechanisms
  207. Incorporation of podoplanin into HIV released from HEK-293T cells, but not PBMC, is required for efficient binding to the attachment factor CLEC-2
  208. Peptide-Based Inhibitors of the HIV Envelope Protein and Other Class I Viral Fusion Proteins
  209. Calcium-modulating cyclophilin ligand does not restrict retrovirus release
  210. Lectin-like interactions in virus–cell recognition
  211. Differential Downregulation of ACE2 by the Spike Proteins of Severe Acute Respiratory Syndrome Coronavirus and Human Coronavirus NL63
  212. Cellular Entry of the SARS Coronavirus: Implications for Transmission, Pathogenicity and Antiviral Strategies
  213. Type II transmembrane serine proteases in cancer and viral infections
  214. Proteolytic Activation of the 1918 Influenza Virus Hemagglutinin
  215. Carbohydrates on HIV: mediators of immune evasion and targets for antiviral intervention
  216. Interactions of LSECtin and DC-SIGN/DC-SIGNR with viral ligands: Differential pH dependence, internalization and virion binding
  217. Analysis of the Interaction of Ebola Virus Glycoprotein with DC‐SIGN (Dendritic Cell–Specific Intercellular Adhesion Molecule 3–Grabbing Nonintegrin) and Its Homologue DC‐SIGNR
  218. A Novel Mechanism for LSECtin Binding to Ebola Virus Surface Glycoprotein through Truncated Glycans
  219. Modulation of HIV and SIV neutralization sensitivity by DC-SIGN and mannose-binding lectin
  220. Discovery and Optimization of a Natural HIV-1 Entry Inhibitor Targeting the gp41 Fusion Peptide
  221. A simian immunodeficiency virus V3 loop mutant that does not efficiently use CCR5 or common alternative coreceptors is moderately attenuated in vivo
  222. The C-type Lectin Receptors CLEC-2 and Dectin-1, but Not DC-SIGN, Signal via a Novel YXXL-dependent Signaling Cascade
  223. Attachment of human immunodeficiency virus to cells and its inhibition
  224. DC-SIGN and CLEC-2 Mediate Human Immunodeficiency Virus Type 1 Capture by Platelets
  225. Modulation of virion incorporation of Ebolavirus glycoprotein: Effects on attachment, cellular entry and neutralization
  226. Highly Conserved Regions within the Spike Proteins of Human Coronaviruses 229E and NL63 Determine Recognition of Their Respective Cellular Receptors
  227. The Signal Peptide of the Ebolavirus Glycoprotein Influences Interaction with the Cellular Lectins DC-SIGN and DC-SIGNR
  228. Cellular Entry of HIV: Evaluation of Therapeutic Targets
  229. Impact of polymorphisms in the DC-SIGNR neck domain on the interaction with pathogens
  230. Functional comparison of mouse CIRE/mouse DC-SIGN and human DC-SIGN
  231. Evidence that multiple defects in murine DC-SIGN inhibit a functional interaction with pathogens
  232. A novel Syk-dependent mechanism of platelet activation by the C-type lectin receptor CLEC-2
  233. Interaction Between the Spike Protein of Human Coronavirus NL63 and its Cellular Receptor ACE2
  234. Attachment Factor and Receptor Engagement of Sars Coronavirus and Human Coronavirus NL63
  235. LSECtin interacts with filovirus glycoproteins and the spike protein of SARS coronavirus
  236. Human coronavirus NL63 employs the severe acute respiratory syndrome coronavirus receptor for cellular entry
  237. DC-SIGN and DC-SIGNR Interact with the Glycoprotein of Marburg Virus and the S Protein of Severe Acute Respiratory Syndrome Coronavirus
  238. Cellular entry of the SARS coronavirus
  239. Differential regulation of human immunodeficiency virus type 2 and simian immunodeficiency virus promoter activity
  240. Prospects of HIV‐1 entry inhibitors as novel therapeutics
  241. Susceptibility to SARS coronavirus S protein-driven infection correlates with expression of angiotensin converting enzyme 2 and infection can be blocked by soluble receptor
  242. S Protein of Severe Acute Respiratory Syndrome-Associated Coronavirus Mediates Entry into Hepatoma Cell Lines and Is Targeted by Neutralizing Antibodies in Infected Patients
  243. Amino Acid 324 in the Simian Immunodeficiency Virus SIVmac V3 Loop Can Confer CD4 Independence and Modulate the Interaction with CCR5 and Alternative Coreceptors
  244. Mutations in the C3 region of human and simian immunodeficiency virus envelope have differential effects on viral infectivity, replication, and CD4-dependency
  245. Hepatitis C Virus Glycoproteins Interact with DC-SIGN and DC-SIGNR
  246. Differential N-Linked Glycosylation of Human Immunodeficiency Virus and Ebola Virus Envelope Glycoproteins Modulates Interactions with DC-SIGN and DC-SIGNR
  247. DC-SIGN and DC-SIGNR Bind Ebola Glycoproteins and Enhance Infection of Macrophages and Endothelial Cells
  248. Sensitivity of HIV-1 to entry inhibitors correlates with envelope/coreceptor affinity, receptor density, and fusion kinetics
  249. Diversity of receptors binding HIV on dendritic cell subsets
  250. Quantitative Expression and Virus Transmission Analysis of DC-SIGN on Monocyte-Derived Dendritic Cells
  251. The role of DC-SIGN and DC-SIGNR in HIV and Ebola virus infection: can potential therapeutics block virus transmission and dissemination?
  252. Hemofiltrate CC Chemokine 1[9-74] Causes Effective Internalization of CCR5 and Is a Potent Inhibitor of R5-Tropic Human Immunodeficiency Virus Type 1 Strains in Primary T Cells and Macrophages
  253. CD4 Independence of Simian Immunodeficiency Virus Envs Is Associated with Macrophage Tropism, Neutralization Sensitivity, and Attenuated Pathogenicity
  254. Evaluation of Current Approaches to Inhibit HIV Entry
  255. Expression of DC-SIGN by Dendritic Cells of Intestinal and Genital Mucosae in Humans and Rhesus Macaques
  256. DC-SIGN and DC-SIGNR: helping hands for HIV
  257. Functional and Antigenic Characterization of Human, Rhesus Macaque, Pigtailed Macaque, and Murine DC-SIGN
  258. DC-SIGN Interactions with Human Immunodeficiency Virus: Virus Binding and Transfer Are Dissociable Functions
  259. Placental expression of DC-SIGN may mediate intrauterine vertical transmission of HIV
  260. The Role of DC-SIGN and DC-SIGNR in HIV and SIV Attachment, Infection, and Transmission
  261. Basic Amino Acid Residues in the V3 Loop of Simian Immunodeficiency Virus Envelope Alter Viral Coreceptor Tropism and Infectivity but Do Not Allow Efficient Utilization of CXCR4 as Entry Cofactor
  262. DC-SIGN Interactions with Human Immunodeficiency Virus Type 1 and 2 and Simian Immunodeficiency Virus
  263. DC-SIGNR, a DC-SIGN homologue expressed in endothelial cells, binds to human and simian immunodeficiency viruses and activates infection in trans
  264. Natural Proteolytic Processing of Hemofiltrate Cc Chemokine 1 Generates a Potent Cc Chemokine Receptor (Ccr)1 and Ccr5 Agonist with Anti-HIV Properties
  265. Simian Immunodeficiency Virus Utilizes Human and Sooty Mangabey but Not Rhesus Macaque STRL33 for Efficient Entry
  266. Co‐receptor Usage of BOB/GPR15 in Addition to CCR5 Has No Significant Effect on Replication of Simian Immunodeficiency Virus In Vivo