All Stories

  1. The conserved QTQTX motif in the SARS-CoV-2 spike protein is dispensable for cleavage and lung cell entry of the emerging variant BA.3.2
  2. JN.1-adapted vaccination is associated with readjustment of ancestral memory B cells toward neutralization within the JN.1 antigenic space
  3. The conserved QTQTX motif in the SARS-CoV-2 spike protein is dispensable for cleavage and lung cell entry of the emerging variant BA.3.2
  4. Lock out: targeting TMPRSS2 to block influenza and coronaviruses
  5. A mutation in the spike protein of MERS-CoV confers resistance against soluble DPP4
  6. Development of immortalized Callithrix jacchus kidney cell lines supporting infection with a panel of viruses
  7. Development of immortalized Callithrix jacchus kidney cell lines supporting infection with a panel of viruses
  8. Amino acid residues 655 and 969 in the spike protein of Omicron subvariant BA.1 control use of TMPRSS2 versus Cathepsin L dependent entry pathways and cell tropism
  9. Polyvalent Mannuronic Acid-Coated Gold Nanoparticles for Probing Multivalent Lectin–Glycan Interaction and Blocking Virus Infection
  10. Simulations and active learning enable efficient identification of an experimentally-validated broad coronavirus inhibitor
  11. Q1020R in the spike proteins of MERS-CoV from Arabian camels and recent human cases confers resistance against soluble human DPP4
  12. Entry Efficiency, Protease Dependence, and Antibody-Mediated Neutralization of SARS-CoV-2 Sublineages KP.3.1.1 and XEC
  13. Immune Response to SARS-CoV-2 XBB.1.5 and JN.1 Variants Following XBB.1.5 Booster Vaccination in Liver Transplant Recipients
  14. Comparative Analysis of Host Cell Entry Efficiency and Neutralization Sensitivity of Emerging SARS-CoV-2 Lineages KP.2, KP.2.3, KP.3, and LB.1
  15. Impact of JN.1 booster vaccination on neutralisation of SARS-CoV-2 variants KP.3.1.1 and XEC
  16. Interferon-Stimulated Genes that Target Retrovirus Translation
  17. Development of rhesus macaque astrocyte cell lines supporting infection with a panel of viruses
  18. Virological Traits of the SARS-CoV-2 BA.2.87.1 Lineage
  19. The Inhibition of Gag-Pol Expression by the Restriction Factor Shiftless Is Dispensable for the Restriction of HIV-1 Infection
  20. Diversification of the VH3‐53 immunoglobulin gene segment by somatic hypermutation results in neutralization of SARS‐CoV‐2 virus variants
  21. Immune responses following BNT162b2 XBB.1.5 vaccination in patients on haemodialysis in Germany
  22. SARS-CoV-2 BA.2.86 enters lung cells and evades neutralizing antibodies with high efficiency
  23. Host cell entry and neutralisation sensitivity of the SARS-CoV-2 XBB.1.16 lineage
  24. Development of immortalized rhesus macaque kidney cells supporting infection with a panel of viruses
  25. Discovery of Polyphenolic Natural Products as SARS-CoV-2 Mpro Inhibitors for COVID-19
  26. TMPRSS2 Is Essential for SARS-CoV-2 Beta and Omicron Infection
  27. Primate Simplexviruses Differ in Tropism for Macaque Cells
  28. Discovery of Polyphenolic Natural Products as SARS-CoV-2 Mpro Inhibitors for COVID-19
  29. The SARS-CoV-2 Delta-Omicron Recombinant Lineage (XD) Exhibits Immune-Escape Properties Similar to the Omicron (BA.1) Variant
  30. Host Cell Entry and Neutralization Sensitivity of SARS-CoV-2 Lineages B.1.620 and R.1
  31. Combinations of Host- and Virus-Targeting Antiviral Drugs Confer Synergistic Suppression of SARS-CoV-2
  32. Publisher Correction: Dalbavancin: novel candidate for COVID-19 treatment
  33. BNT162b2-boosted immune responses six months after heterologous or homologous ChAdOx1nCoV-19/BNT162b2 vaccination against COVID-19
  34. BNT162b2 booster after heterologous prime-boost vaccination induces potent neutralizing antibodies and T cell reactivity against SARS-CoV-2 Omicron BA.1 in young adults
  35. Nafamostat-Mediated Inhibition of SARS-CoV-2 Ribosomal Frameshifting Is Insufficient to Impair Viral Replication in Vero Cells. Comment on Munshi et al. Identifying Inhibitors of −1 Programmed Ribosomal Frameshifting in a Broad Spectrum of Coronaviruse...
  36. Peptidomimetic inhibitors of TMPRSS2 block SARS-CoV-2 infection in cell culture
  37. Mutagenic Analysis of the HIV Restriction Factor Shiftless
  38. Evidence for an ACE2-Independent Entry Pathway That Can Protect from Neutralization by an Antibody Used for COVID-19 Therapy
  39. Small-Molecule Thioesters as SARS-CoV-2 Main Protease Inhibitors: Enzyme Inhibition, Structure–Activity Relationships, Antiviral Activity, and X-ray Structure Determination
  40. Comparable neutralisation evasion of SARS-CoV-2 omicron subvariants BA.1, BA.2, and BA.3
  41. Efficient antibody evasion but reduced ACE2 binding by the emerging SARS-CoV-2 variant B.1.640.2
  42. Understanding Omicron: Transmissibility, immune evasion and antiviral intervention
  43. SARS-CoV-2 Omicron sublineages show comparable cell entry but differential neutralization by therapeutic antibodies
  44. SARS-CoV-2 variants C.1.2 and B.1.621 (Mu) partially evade neutralization by antibodies elicited upon infection or vaccination
  45. Investigations on SARS-CoV-2 Susceptibility of Domestic and Wild Animals Using Primary Cell Culture Models Derived from the Upper and Lower Respiratory Tract
  46. Omicron: Master of immune evasion maintains robust ACE2 binding
  47. Functional analysis of polymorphisms at the S1/S2 site of SARS-CoV-2 spike protein
  48. Augmented neutralization of SARS‐CoV‐2 Omicron variant by boost vaccination and monoclonal antibodies
  49. Neutralizing antibody responses 300 days after SARS‐CoV‐2 infection and induction of high antibody titers after vaccination
  50. Alternatives to animal models and their application in the discovery of species susceptibility to SARS-CoV-2 and other respiratory infectious pathogens: A review
  51. The Omicron variant is highly resistant against antibody-mediated neutralization: Implications for control of the COVID-19 pandemic
  52. Rapid SARS-CoV-2 Adaptation to Available Cellular Proteases
  53. MCMV-based vaccine vectors expressing full-length viral proteins provide long-term humoral immune protection upon a single-shot vaccination
  54. No evidence for increased cell entry or antibody evasion by Delta sublineage AY.4.2
  55. A Recombinant System and Reporter Viruses for Papiine Alphaherpesvirus 2
  56. The MEK1/2-inhibitor ATR-002 efficiently blocks SARS-CoV-2 propagation and alleviates pro-inflammatory cytokine/chemokine responses
  57. Heterologous ChAdOx1 nCoV-19 and BNT162b2 prime-boost vaccination elicits potent neutralizing antibody responses and T cell reactivity against prevalent SARS-CoV-2 variants
  58. Activation of Sphingomyelinase-Ceramide-Pathway in COVID-19 Purposes Its Inhibition for Therapeutic Strategies
  59. Novel SARS-CoV-2 receptors: ASGR1 and KREMEN1
  60. Erythrocytes increase endogenous sphingosine 1-phosphate levels as an adaptive response to SARS-CoV-2 infection
  61. Improved cellular and humoral immunity upon a second BNT162b2 and mRNA-1273 boost in prime-boost vaccination no/low responders with end-stage renal disease
  62. Protective mucosal immunity against SARS-CoV-2 after heterologous systemic prime-mucosal boost immunization
  63. Spike residue 403 affects binding of coronavirus spikes to human ACE2
  64. The spike protein of SARS-CoV-2 variant A.30 is heavily mutated and evades vaccine-induced antibodies with high efficiency
  65. Evidence that two instead of one defective interfering RNA in influenza A virus-derived defective interfering particles (DIPs) does not enhance antiviral activity
  66. A novel class of TMPRSS2 inhibitors potently block SARS-CoV-2 and MERS-CoV viral entry and protect human epithelial lung cells
  67. Delta variant (B.1.617.2) sublineages do not show increased neutralization resistance
  68. B.1.617.2 enters and fuses lung cells with increased efficiency and evades antibodies induced by infection and vaccination
  69. The Upper Respiratory Tract of Felids Is Highly Susceptible to SARS-CoV-2 Infection
  70. A pair of noncompeting neutralizing human monoclonal antibodies protecting from disease in a SARS‐CoV‐2 infection model
  71. Functional comparison of MERS-coronavirus lineages reveals increased replicative fitness of the recombinant lineage 5
  72. SARS-CoV-2 delta variant neutralisation after heterologous ChAdOx1-S/BNT162b2 vaccination
  73. Neutralization of the SARS-CoV-2 Delta variant after heterologous and homologous BNT162b2 or ChAdOx1 nCoV-19 vaccination
  74. Immune responses against SARS-CoV-2 variants after heterologous and homologous ChAdOx1 nCoV-19/BNT162b2 vaccination
  75. SARS-CoV-2 neutralizing antibodies: Longevity, breadth, and evasion by emerging viral variants
  76. SARS-CoV-2 variant B.1.617 is resistant to bamlanivimab and evades antibodies induced by infection and vaccination
  77. How SARS-CoV-2 makes the cut
  78. Humoral and Cellular Immune Responses Against Severe Acute Respiratory Syndrome Coronavirus 2 Variants and Human Coronaviruses After Single BNT162b2 Vaccination
  79. Urinary Levels of SARS-CoV-2 Nucleocapsid Protein Associate With Risk of AKI and COVID-19 Severity: A Single-Center Observational Study
  80. Therapeutic Application of alpha-1-antitrypsin in COVID-19
  81. Cell culture-based production and in vivo characterization of purely clonal defective interfering influenza virus particles
  82. The SARS-CoV-2 and other human coronavirus spike proteins are fine-tuned towards temperature and proteases of the human airways
  83. SARS-CoV-2 mutations acquired in mink reduce antibody-mediated neutralization
  84. The sphingosine kinase 1 activator, K6PC-5, attenuates Ebola virus infection
  85. SARS-CoV-2 variants B.1.351 and P.1 escape from neutralizing antibodies
  86. Alpha-1 antitrypsin inhibits TMPRSS2 protease activity and SARS-CoV-2 infection
  87. Ex vivo assay to evaluate the efficacy of drugs targeting sphingolipids in preventing SARS-CoV-2 infection of nasal epithelial cells
  88. Mutation D614G increases SARS-CoV-2 transmission
  89. Camostat mesylate inhibits SARS-CoV-2 activation by TMPRSS2-related proteases and its metabolite GBPA exerts antiviral activity
  90. Dalbavancin: novel candidate for COVID-19 treatment
  91. Natural cystatin C fragments inhibit GPR15-mediated HIV and SIV infection without interfering with GPR15L signaling
  92. Molecular mechanism of inhibiting the SARS-CoV-2 cell entry facilitator TMPRSS2 with camostat and nafamostat
  93. Inhibition of acid sphingomyelinase by ambroxol prevents SARS-CoV-2 entry into epithelial cells
  94. Synergistic inhibition of SARS-CoV-2 cell entry by otamixaban and covalent protease inhibitors: pre-clinical assessment of pharmacological and molecular properties
  95. The SARS-CoV-2 and other human coronavirus spike proteins are fine-tuned towards temperature and proteases of the human airways
  96. Low serum neutralizing anti-SARS-CoV-2 S antibody levels in mildly affected COVID-19 convalescent patients revealed by two different detection methods
  97. Corrigendum: Compact, Polyvalent Mannose Quantum Dots as Sensitive, Ratiometric FRET Probes for Multivalent Protein–Ligand Interactions
  98. Camostat Mesylate May Reduce Severity of Coronavirus Disease 2019 Sepsis: A First Observation
  99. Pharmacological Inhibition of Acid Sphingomyelinase Prevents Uptake of SARS-CoV-2 by Epithelial Cells
  100. Sphingosine prevents binding of SARS–CoV-2 spike to its cellular receptor ACE2
  101. Glycan-Gold Nanoparticles as Multifunctional Probes for Multivalent Lectin–Carbohydrate Binding: Implications for Blocking Virus Infection and Nanoparticle Assembly
  102. LY6E impairs coronavirus fusion and confers immune control of viral disease
  103. Chloroquine does not inhibit SARS-CoV-2
  104. Probing Multivalent Lectin-Carbohydrate Binding via Multifunctional Glycan-Gold Nanoparticles: Implications for Blocking Virus Infection
  105. Probing Multivalent Lectin-Carbohydrate Binding via Multifunctional Glycan-Gold Nanoparticles: Implications for Blocking Virus Infection
  106. Furin cleavage of SARS-CoV-2 spike is required for infection of lung cells
  107. Structural Basis for Potent Neutralization of Betacoronaviruses by Single-Domain Camelid Antibodies
  108. H2 influenza A virus is not pathogenic in Tmprss2 knock-out mice
  109. Nafamostat inhibits SARS-CoV-2
  110. SARS-CoV-2 uses ACE2 and TMPRSS2 for infection of lung cells
  111. SARS-CoV-2 uses ACE2 and TMPRSS2 for infection of lung cells
  112. Polymorphisms in dipeptidyl peptidase 4 reduce host cell entry of Middle East respiratory syndrome coronavirus
  113. A Fosmid-Based System for the Generation of Recombinant Cercopithecine Alphaherpesvirus 2 Encoding Reporter Genes
  114. Role of rhesus macaque IFITM3(2) in simian immunodeficiency virus infection of macaques
  115. Analysis of Resistance of Ebola Virus Glycoprotein-Driven Entry Against MDL28170, An Inhibitor of Cysteine Cathepsins
  116. Hemagglutinin Cleavability, Acid Stability, and Temperature Dependence Optimize Influenza B Virus for Replication in Human Airways
  117. Spike proteins of novel MERS-coronavirus isolates from North- and West-African dromedary camels mediate robust viral entry into human target cells
  118. Evidence for influenza B virus hemagglutinin adaptation to the human host: high cleavability, acid-stability and preference for cool temperature
  119. Analysis of IFITM-IFITM Interactions by a Flow Cytometry-Based FRET Assay
  120. Kaposi sarcoma in a guereza monkey
  121. Novel Virus Related to Kaposi’s Sarcoma–Associated Herpesvirus from Colobus Monkey
  122. Evidence that Calu-3 cells are largely resistant to Ebola virus entry
  123. Tmprss2 knock-out mice are resistant to H10 influenza A virus pathogenesis
  124. IFITM s mediate viral evasion in acute and chronic hepatitis C virus infection
  125. Guanylate-Binding Proteins 2 and 5 Exert Broad Antiviral Activity by Inhibiting Furin-Mediated Processing of Viral Envelope Proteins
  126. SPP and SKI-1 inhibitor block Ebola virus entry
  127. Modulation of HIV-1 Gag/Gag-Pol frameshifting by tRNA abundance
  128. Detection of antibodies against viruses in a macaque colony using a chip-based approach
  129. The cell Line SH-SY5Y is resistant to infection by Ebola virus and other filoviruses
  130. A safe system for production of defective interfering particles
  131. Virosomes interfere with Ebola virus control by the immune system
  132. Focal epithelial hyperplasia and papillomavirus infection in a bonobo
  133. Development and use of Lentiviral Vectors Pseudotyped with Influenza B Haemagglutinins: application to vaccine immunogenicity, mAb potency and sero-surveillance studies
  134. Tetherin efficiently inhibits Nipah virus but not Ebola virus spread in fruit bat cells
  135. MERS-coronavirus spike protein: Importance of protease cleavage sites for viral entry
  136. A MERS-coronavirus variant that is partially resistant against neutralizing antibodies
  137. The cellular protease TMPRSS11A can convert influenza virus into an infectious form
  138. Cell Entry of Influenza A Viruses: Sweet Talk between HA and Ca V 1.2
  139. A GXXXA motif in the Ebola virus glycoprotein is required for tetherin antagonism
  140. Attachment/Binding
  141. Priming Time: How Cellular Proteases Arm Coronavirus Spike Proteins
  142. VSV-G antagonizes tetherin in transfected cells
  143. Inhibition of lectin-dependent enhancement of Ebola virus entry into cells
  144. Identification of sites in SARS-S required cleavage and activation by TMPRSS2
  145. TMPRSS2 of non-human primates activates influenza viruses
  146. Herpes B virus replication and viral lesions in the liver of a cynomolgus macaque which died from severe disease with rapid onset
  147. pH Optimum of Hemagglutinin-Mediated Membrane Fusion Determines Sensitivity of Influenza A Viruses to the Interferon-Induced Antiviral State and IFITMs
  148. Rhesus macaque IFITM3 gene polymorphisms and SIV infection
  149. A Polymorphism within the Internal Fusion Loop of the Ebola Virus Glycoprotein Modulates Host Cell Entry
  150. Detection systems for antibody responses against herpes B virus
  151. Evidence that Processing of the Severe Fever with Thrombocytopenia Syndrome Virus Gn/Gc Polyprotein Is Critical for Viral Infectivity and Requires an Internal Gc Signal Peptide
  152. Virion Background and Efficiency of Virion Incorporation Determine Susceptibility of Simian Immunodeficiency Virus Env-Driven Viral Entry to Inhibition by IFITM Proteins
  153. The Tetherin Antagonism of the Ebola Virus Glycoprotein Requires an Intact Receptor-Binding Domain and Can Be Blocked by GP1-Specific Antibodies
  154. The Role of Phlebovirus Glycoproteins in Viral Entry, Assembly and Release
  155. The Hemagglutinin of Bat-Associated Influenza Viruses Is Activated by TMPRSS2 for pH-Dependent Entry into Bat but Not Human Cells
  156. Compact, Polyvalent Mannose Quantum Dots as Sensitive, Ratiometric FRET Probes for Multivalent Protein-Ligand Interactions
  157. Compact, Polyvalent Mannose Quantum Dots as Sensitive, Ratiometric FRET Probes for Multivalent Protein-Ligand Interactions
  158. The Glycoproteins of All Filovirus Species Use the Same Host Factors for Entry into Bat and Human Cells but Entry Efficiency Is Species Dependent
  159. The Proteolytic Activation of (H3N2) Influenza A Virus Hemagglutinin Is Facilitated by Different Type II Transmembrane Serine Proteases
  160. TMPRSS2 Isoform 1 Activates Respiratory Viruses and Is Expressed in Viral Target Cells
  161. Exclusive Decoration of Simian Immunodeficiency Virus Env with High-Mannose Type N-Glycans Is Not Compatible with Mucosal Transmission in Rhesus Macaques
  162. Tetherin Sensitivity of Influenza A Viruses Is Strain Specific: Role of Hemagglutinin and Neuraminidase
  163. Interferon-Induced Transmembrane Protein–Mediated Inhibition of Host Cell Entry of Ebolaviruses
  164. Analysis of Ebola Virus Entry Into Macrophages
  165. Comparative Analysis of Host Cell Entry of Ebola Virus From Sierra Leone, 2014, and Zaire, 1976
  166. Protease inhibitors targeting coronavirus and filovirus entry
  167. IFITM Proteins Inhibit Entry Driven by the MERS-Coronavirus Spike Protein: Evidence for Cholesterol-Independent Mechanisms
  168. DESC1 and MSPL Activate Influenza A Viruses and Emerging Coronaviruses for Host Cell Entry
  169. Inhibition of Proprotein Convertases Abrogates Processing of the Middle Eastern Respiratory Syndrome Coronavirus Spike Protein in Infected Cells but Does Not Reduce Viral Infectivity
  170. Bitter-sweet symphony: glycan–lectin interactions in virus biology
  171. Influenza A Virus Encoding Secreted Gaussia Luciferase as Useful Tool to Analyze Viral Replication and Its Inhibition by Antiviral Compounds and Cellular Proteins
  172. Analysis of Determinants in Filovirus Glycoproteins Required for Tetherin Antagonism
  173. The clinically approved drugs amiodarone, dronedarone and verapamil inhibit filovirus cell entry
  174. Induced and spontaneous heart rate turbulence in mice: influence of coupling interval
  175. Toll-Like Receptor 3 Signalling Up-Regulates Expression of the HIV Co-Receptor G-Protein Coupled Receptor 15 on Human CD4+ T Cells
  176. Tmprss2 Is Essential for Influenza H1N1 Virus Pathogenesis in Mice
  177. Lack of MERS Coronavirus Neutralizing Antibodies in Humans, Eastern Province, Saudi Arabia
  178. Proteolytic activation of the SARS-coronavirus spike protein: Cutting enzymes at the cutting edge of antiviral research
  179. TMPRSS2 and ADAM17 Cleave ACE2 Differentially and Only Proteolysis by TMPRSS2 Augments Entry Driven by the Severe Acute Respiratory Syndrome Coronavirus Spike Protein
  180. Platelet activation suppresses HIV-1 infection of T cells
  181. TMPRSS2 Activates the Human Coronavirus 229E for Cathepsin-Independent Host Cell Entry and Is Expressed in Viral Target Cells in the Respiratory Epithelium
  182. The Spike Protein of the Emerging Betacoronavirus EMC Uses a Novel Coronavirus Receptor for Entry, Can Be Activated by TMPRSS2, and Is Targeted by Neutralizing Antibodies
  183. Severe Fever with Thrombocytopenia Virus Glycoproteins Are Targeted by Neutralizing Antibodies and Can Use DC-SIGN as a Receptor for pH-Dependent Entry into Human and Animal Cell Lines
  184. Cellular Entry of Retroviruses
  185. Attachment/Binding
  186. Host Cell Factors in Filovirus Entry: Novel Players, New Insights
  187. The role of the alternative coreceptor GPR15 in SIV tropism for human cells
  188. How Ebola Virus Counters the Interferon System
  189. Influenza A Virus Does Not Encode a Tetherin Antagonist with Vpu-Like Activity and Induces IFN-Dependent Tetherin Expression in Infected Cells
  190. Influenza and SARS-Coronavirus Activating Proteases TMPRSS2 and HAT Are Expressed at Multiple Sites in Human Respiratory and Gastrointestinal Tracts
  191. Cathepsins B and L activate Ebola but not Marburg virus glycoproteins for efficient entry into cell lines and macrophages independent of TMPRSS2 expression
  192. CD4- and dynamin-dependent endocytosis of HIV-1 into plasmacytoid dendritic cells
  193. Comparative Analysis of Ebola Virus Glycoprotein Interactions With Human and Bat Cells
  194. The Ebola Virus Glycoprotein and HIV-1 Vpu Employ Different Strategies to Counteract the Antiviral Factor Tetherin
  195. The SARS-Coronavirus-Host Interactome: Identification of Cyclophilins as Target for Pan-Coronavirus Inhibitors
  196. Cleavage and Activation of the Severe Acute Respiratory Syndrome Coronavirus Spike Protein by Human Airway Trypsin-Like Protease
  197. DC-SIGN: Access Portal for Sweet Viral Killers
  198. Different host cell proteases activate the SARS-coronavirus spike-protein for cell–cell and virus–cell fusion
  199. Evidence that TMPRSS2 Activates the Severe Acute Respiratory Syndrome Coronavirus Spike Protein for Membrane Fusion and Reduces Viral Control by the Humoral Immune Response
  200. Mouse LSECtin as a model for a human Ebola virus receptor
  201. The multiple facets of HIV attachment to dendritic cell lectins
  202. Novel insights into proteolytic cleavage of influenza virus hemagglutinin
  203. TMPRSS2 and TMPRSS4 Facilitate Trypsin-Independent Spread of Influenza Virus in Caco-2 Cells
  204. A Single Asparagine-Linked Glycosylation Site of the Severe Acute Respiratory Syndrome Coronavirus Spike Glycoprotein Facilitates Inhibition by Mannose-Binding Lectin through Multiple Mechanisms
  205. Incorporation of podoplanin into HIV released from HEK-293T cells, but not PBMC, is required for efficient binding to the attachment factor CLEC-2
  206. Peptide-Based Inhibitors of the HIV Envelope Protein and Other Class I Viral Fusion Proteins
  207. Calcium-modulating cyclophilin ligand does not restrict retrovirus release
  208. Lectin-like interactions in virus–cell recognition
  209. Differential Downregulation of ACE2 by the Spike Proteins of Severe Acute Respiratory Syndrome Coronavirus and Human Coronavirus NL63
  210. Cellular Entry of the SARS Coronavirus: Implications for Transmission, Pathogenicity and Antiviral Strategies
  211. Type II transmembrane serine proteases in cancer and viral infections
  212. Proteolytic Activation of the 1918 Influenza Virus Hemagglutinin
  213. Carbohydrates on HIV: mediators of immune evasion and targets for antiviral intervention
  214. Interactions of LSECtin and DC-SIGN/DC-SIGNR with viral ligands: Differential pH dependence, internalization and virion binding
  215. Analysis of the Interaction of Ebola Virus Glycoprotein with DC‐SIGN (Dendritic Cell–Specific Intercellular Adhesion Molecule 3–Grabbing Nonintegrin) and Its Homologue DC‐SIGNR
  216. A Novel Mechanism for LSECtin Binding to Ebola Virus Surface Glycoprotein through Truncated Glycans
  217. Modulation of HIV and SIV neutralization sensitivity by DC-SIGN and mannose-binding lectin
  218. Discovery and Optimization of a Natural HIV-1 Entry Inhibitor Targeting the gp41 Fusion Peptide
  219. A simian immunodeficiency virus V3 loop mutant that does not efficiently use CCR5 or common alternative coreceptors is moderately attenuated in vivo
  220. The C-type Lectin Receptors CLEC-2 and Dectin-1, but Not DC-SIGN, Signal via a Novel YXXL-dependent Signaling Cascade
  221. Attachment of human immunodeficiency virus to cells and its inhibition
  222. DC-SIGN and CLEC-2 Mediate Human Immunodeficiency Virus Type 1 Capture by Platelets
  223. Modulation of virion incorporation of Ebolavirus glycoprotein: Effects on attachment, cellular entry and neutralization
  224. Highly Conserved Regions within the Spike Proteins of Human Coronaviruses 229E and NL63 Determine Recognition of Their Respective Cellular Receptors
  225. The Signal Peptide of the Ebolavirus Glycoprotein Influences Interaction with the Cellular Lectins DC-SIGN and DC-SIGNR
  226. Cellular Entry of HIV: Evaluation of Therapeutic Targets
  227. Impact of polymorphisms in the DC-SIGNR neck domain on the interaction with pathogens
  228. Functional comparison of mouse CIRE/mouse DC-SIGN and human DC-SIGN
  229. Evidence that multiple defects in murine DC-SIGN inhibit a functional interaction with pathogens
  230. A novel Syk-dependent mechanism of platelet activation by the C-type lectin receptor CLEC-2
  231. Interaction Between the Spike Protein of Human Coronavirus NL63 and its Cellular Receptor ACE2
  232. Attachment Factor and Receptor Engagement of Sars Coronavirus and Human Coronavirus NL63
  233. LSECtin interacts with filovirus glycoproteins and the spike protein of SARS coronavirus
  234. Human coronavirus NL63 employs the severe acute respiratory syndrome coronavirus receptor for cellular entry
  235. DC-SIGN and DC-SIGNR Interact with the Glycoprotein of Marburg Virus and the S Protein of Severe Acute Respiratory Syndrome Coronavirus
  236. Cellular entry of the SARS coronavirus
  237. Differential regulation of human immunodeficiency virus type 2 and simian immunodeficiency virus promoter activity
  238. Prospects of HIV‐1 entry inhibitors as novel therapeutics
  239. Susceptibility to SARS coronavirus S protein-driven infection correlates with expression of angiotensin converting enzyme 2 and infection can be blocked by soluble receptor
  240. S Protein of Severe Acute Respiratory Syndrome-Associated Coronavirus Mediates Entry into Hepatoma Cell Lines and Is Targeted by Neutralizing Antibodies in Infected Patients
  241. Amino Acid 324 in the Simian Immunodeficiency Virus SIVmac V3 Loop Can Confer CD4 Independence and Modulate the Interaction with CCR5 and Alternative Coreceptors
  242. Mutations in the C3 region of human and simian immunodeficiency virus envelope have differential effects on viral infectivity, replication, and CD4-dependency
  243. Hepatitis C Virus Glycoproteins Interact with DC-SIGN and DC-SIGNR
  244. Differential N-Linked Glycosylation of Human Immunodeficiency Virus and Ebola Virus Envelope Glycoproteins Modulates Interactions with DC-SIGN and DC-SIGNR
  245. DC-SIGN and DC-SIGNR Bind Ebola Glycoproteins and Enhance Infection of Macrophages and Endothelial Cells
  246. Sensitivity of HIV-1 to entry inhibitors correlates with envelope/coreceptor affinity, receptor density, and fusion kinetics
  247. Diversity of receptors binding HIV on dendritic cell subsets
  248. Quantitative Expression and Virus Transmission Analysis of DC-SIGN on Monocyte-Derived Dendritic Cells
  249. The role of DC-SIGN and DC-SIGNR in HIV and Ebola virus infection: can potential therapeutics block virus transmission and dissemination?
  250. Hemofiltrate CC Chemokine 1[9-74] Causes Effective Internalization of CCR5 and Is a Potent Inhibitor of R5-Tropic Human Immunodeficiency Virus Type 1 Strains in Primary T Cells and Macrophages
  251. CD4 Independence of Simian Immunodeficiency Virus Envs Is Associated with Macrophage Tropism, Neutralization Sensitivity, and Attenuated Pathogenicity
  252. Evaluation of Current Approaches to Inhibit HIV Entry
  253. Expression of DC-SIGN by Dendritic Cells of Intestinal and Genital Mucosae in Humans and Rhesus Macaques
  254. DC-SIGN and DC-SIGNR: helping hands for HIV
  255. Functional and Antigenic Characterization of Human, Rhesus Macaque, Pigtailed Macaque, and Murine DC-SIGN
  256. DC-SIGN Interactions with Human Immunodeficiency Virus: Virus Binding and Transfer Are Dissociable Functions
  257. Placental expression of DC-SIGN may mediate intrauterine vertical transmission of HIV
  258. The Role of DC-SIGN and DC-SIGNR in HIV and SIV Attachment, Infection, and Transmission
  259. Basic Amino Acid Residues in the V3 Loop of Simian Immunodeficiency Virus Envelope Alter Viral Coreceptor Tropism and Infectivity but Do Not Allow Efficient Utilization of CXCR4 as Entry Cofactor
  260. DC-SIGN Interactions with Human Immunodeficiency Virus Type 1 and 2 and Simian Immunodeficiency Virus
  261. DC-SIGNR, a DC-SIGN homologue expressed in endothelial cells, binds to human and simian immunodeficiency viruses and activates infection in trans
  262. Natural Proteolytic Processing of Hemofiltrate Cc Chemokine 1 Generates a Potent Cc Chemokine Receptor (Ccr)1 and Ccr5 Agonist with Anti-HIV Properties
  263. Simian Immunodeficiency Virus Utilizes Human and Sooty Mangabey but Not Rhesus Macaque STRL33 for Efficient Entry
  264. Co‐receptor Usage of BOB/GPR15 in Addition to CCR5 Has No Significant Effect on Replication of Simian Immunodeficiency Virus In Vivo