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  1. Impaired prolactin actions mediate altered offspring metabolism induced by maternal high-fat feeding during lactation
  2. Remarks on the Prolactin Hypothesis of Peripartum Cardiomyopathy
  3. Chapter 14. From Reproductive Neuroendocrinology and Lactation to Vasoinhibins and Angiogenesis
  4. Vasoinhibin Serum Levels Are Required to Demonstrate Their Role in Peripartum Cardiomyopathy Etiopathology
  5. Prolactin Promotes Adipose Tissue Fitness and Insulin Sensitivity in Obese Males
  6. The vasoinhibin solution structure appears unfolded, dynamic, and features aggregation
  7. Serum proteases do not cleave prolactin to vasoinhibins at physiological pH
  8. Vasoinhibins are natural inhibitors of angiogenesis in the vitreous and are impaired in patients with diabetic retinopathy
  9. Prolactin and blood vessels: A comparative endocrinology perspective
  10. Peptide Hormone Regulation of Angiogenesis
  11. Vasoinhibins: endogenous regulators of angiogenesis and vascular function
  12. Potentiation of Prolactin Secretion following Lactotrope Escape from Dopamine Action
  13. Transient Dopamine Withdrawal Differentially Potentiates Thyrotropin Releasing Hormone-Induced Release of Prolactin of Various Ages
  14. Effect of Dopamine Withdrawal on Activation of Adenylate Cyclase and Phospholipase C in Enriched Lactotrophs*