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  1. Methods to Evaluate the Effects of HAT/KAT Inhibition on SIAH2-Driven Reactive Oxygen Species Generation in Helicobacter pylori-Infected Gastric Epithelial Cells
  2. Editorial: Microbial sensing to control host immune responses
  3. CTK7A, a curcumin derivative, can be a potential candidate for targeting HIF-1α/p300 complex: Evidences from in vitro and computational studies
  4. Hypoxia-driven metabolic heterogeneity and immune evasive behaviour of gastrointestinal cancers: Elements of a recipe for disaster
  5. Siah2–GRP78 interaction regulates ROS and provides a proliferative advantage to Helicobacter pylori-infected gastric epithelial cancer cells
  6. Abstract 5680: Hypoxia-responsive and HIF1α-regulated AAA+ ATPase ATAD2 shows high oncogenic potential in stomach cancer
  7. Silent hypoxia in COVID-19: a gut microbiota connection
  8. Abstract 1988: Association between CEACAMs and hypoxia defines gastric cancer progression
  9. Helicobacter pylori-induced gastric cancer is orchestrated by MRCKβ-mediated Siah2 phosphorylation
  10. Oncogenic potential of ATAD2 in stomach cancer and insights into the protein-protein interactions at its AAA + ATPase domain and bromodomain
  11. HIF1α-dependent upregulation of ATAD2 promotes proliferation and migration of stomach cancer cells in response to hypoxia
  12. Acetylation-mediated Siah2 stabilization enhances PHD3 degradation in Helicobacter pylori–infected gastric epithelial cancer cells
  13. Mechanism of gastric cancer invasiveness
  14. Membrane-bound β-catenin degradation is enhanced by ETS2-mediated Siah1 induction in Helicobacter pylori-infected gastric cancer cells
  15. Inhibition of histone/lysine acetyltransferase activity kills CoCl2-treated and hypoxia-exposed gastric cancer cells and reduces their invasiveness
  16. ETS2 and Twist1 promote invasiveness of Helicobacter pylori-infected gastric cancer cells by inducing Siah2
  17. Cobalt chloride-mediated protein kinase Cα (PKCα) phosphorylation induces hypoxia-inducible factor 1α (HIF1α) in the nucleus of gastric cancer cell
  18. Regulation of Rac1 and Reactive Oxygen Species Production in Response to Infection of Gastrointestinal Epithelia
  19. Mechanisms of apoptosis inhibition in Chlamydia pneumoniae-infected neutrophils
  20. Noxa phosphorylated at S13 is not apoptotic
  21. Oxidative Stress: An Essential Factor in the Pathogenesis of Gastrointestinal Mucosal Diseases
  22. Infection of neutrophil granulocytes with Leishmania major activates ERK 1/2 and modulates multiple apoptotic pathways to inhibit apoptosis
  23. Infection with Anaplasma phagocytophilum Activates the Phosphatidylinositol 3-Kinase/Akt and NF-κB Survival Pathways in Neutrophil Granulocytes
  24. Mechanism of hypoxia-inducible factor 1α-mediated Mcl1 regulation in Helicobacter pylori-infected human gastric epithelium
  25. 651 Apurinic/Apyrimidinic Endonuclease (APE-1) Regulates Oxidative Signaling Pathways Including RAC1 and NADPH Oxidase in H. pylori (HP) Infected Gastric Epithelial Cells (GEC)
  26. Acetylation of Apurinic/Apyrimidinic Endonuclease-1 Regulates Helicobacterpylori-Mediated Gastric Epithelial Cell Apoptosis
  27. Tumor necrosis factor (TNF)-α-induced IL-8 expression in gastric epithelial cells: Role of reactive oxygen species and AP endonuclease-1/redox factor (Ref)-1
  28. Host Immunity in the Development of Gastric Preneoplasia
  29. S1603 Apurinic/Apyrimidinic Endonuclease (APE)-1/Redox Factor (Ref)-1 Enhances Hypoxia Inducible Factor (HIF)-1α Activation in H. pylori-Infected Gastric Epithelium But Inhibits HIF-1α-Mediated Apoptotic Events
  30. 29 Apurinic/Apyrimidinic Endonuclease (APE)-1/Redox Factor (Ref)-1 Is Involved in Gastric Epithelial Cell Survival After H. pylori Infection
  31. Mycobacterium avium-induced matrix metalloproteinase-9 expression occurs in a cyclooxygenase-2-dependent manner and involves phosphorylation- and acetylation-dependent chromatin modification
  32. Direct extracellular interaction between the early secreted antigen ESAT-6 of Mycobacterium tuberculosis and TLR2 inhibits TLR signaling in macrophages
  33. O323 Execution of macrophage apoptosis by PE_PGRS33 of Mycobacterium tuberculosis
  34. Execution of Macrophage Apoptosis by PE_PGRS33 ofMycobacterium tuberculosisIs Mediated by Toll-like Receptor 2-dependent Release of Tumor Necrosis Factor-α
  35. Mycobacterium tuberculosisLipoarabinomannan-mediated IRAK-M Induction Negatively Regulates Toll-like Receptor-dependent Interleukin-12 p40 Production in Macrophages
  36. NF-κB- and C/EBPβ-driven Interleukin-1β Gene Expression and PAK1-mediated Caspase-1 Activation Play Essential Roles in Interleukin-1β Release fromHelicobacter pyloriLipopolysaccharide-stimulated Macrophages
  37. Toll-like Receptor 2 and Mitogen- and Stress-activated Kinase 1 Are Effectors ofMycobacterium avium-induced Cyclooxygenase-2 Expression in Macrophages
  38. Caspase 3-mediated Proteolysis of the N-terminal Cytoplasmic Domain of the Human Erythroid Anion Exchanger 1 (Band 3)
  39. Execution of Macrophage Apoptosis byMycobacterium aviumthrough Apoptosis Signal-regulating Kinase 1/p38 Mitogen-activated Protein Kinase Signaling and Caspase 8 Activation
  40. Mitogen-activated protein kinases and nuclear factor-κB regulate Helicobacter pylori-mediated interleukin-8 release from macrophages
  41. Lipoarabinomannan fromMycobacterium tuberculosisPromotes Macrophage Survival by Phosphorylating Bad through a Phosphatidylinositol 3-Kinase/Akt Pathway