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Vasoconstrictive hormones unmask TRPV1-mediated constrictions of coronary rings. Since capsaicin-induced, endothelium-dependent vasodilatory actions may reduce the absolute strength of capsaicin-induced contractions, in coronary arteries with impaired endothelium function, capsaicin-induced contractions may be exacerbated under proinflammatory conditions, possibly leading to coronary spasm that is known to worsen the outcomes of myocardial infarction and/or post-myocardial infarction recovery
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This page is a summary of: Mechanisms underlying capsaicin effects in canine coronary artery: implications for coronary spasm, Cardiovascular Research, June 2014, European Society of Cardiology,
DOI: 10.1093/cvr/cvu152.
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