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The molecular mechanisms of initial response to ischemic insult in the heart and the pathways involved in compensation and remodeling are still largely unknown. Authors identify a significant protective effect of Protein disulfide isomerase (PDI), an endoplasmic reticulum enzyme responsible for protein folding, in human infarcted hearts mediated by a reduction of apoptotic rate and by prevention of cardiac remodeling
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This page is a summary of: Identification of Protein Disulfide Isomerase as a Cardiomyocyte Survival Factor in Ischemic Cardiomyopathy, Journal of the American College of Cardiology, September 2007, Elsevier,
DOI: 10.1016/j.jacc.2007.06.006.
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