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  1. The Neisseria gonorrhoeae type IV pilus promotes resistance to hydrogen peroxide- and LL-37-mediated killing by modulating the availability of intracellular, labile iron
  2. Transcriptional initiation of a small RNA, not R‐loop stability, dictates the frequency of pilin antigenic variation inNeisseria gonorrhoeae
  3. Neisseria gonorrhoeae Exposed to Sublethal Levels of Hydrogen Peroxide Mounts a Complex Transcriptional Response
  4. Analysis of Pilin Antigenic Variation in Neisseria meningitidis by Next-Generation Sequencing
  5. The low-molecular-mass, penicillin-binding proteins DacB and DacC combine to modify peptidoglycan cross-linking and allow stable Type IV pilus expression in Neisseria gonorrhoeae
  6. Analyzing Neisseria gonorrhoeae Pilin Antigenic Variation Using 454 Sequencing Technology
  7. Seminal Plasma Promotes Neisseria gonorrhoeae Aggregation and Biofilm Formation
  8. The Gonococcal NlpD Protein Facilitates Cell Separation by Activating Peptidoglycan Cleavage by AmiC
  9. Neisseria gonorrhoeae MutS Affects Pilin Antigenic Variation through Mismatch Correction and Not bypilEGuanine Quartet Binding
  10. Neisseria gonorrhoeae Elicits Extracellular Traps in Primary Neutrophil Culture While Suppressing the Oxidative Burst
  11. Seminal Plasma Initiates a Neisseria gonorrhoeae Transmission State
  12. Saturating Mutagenesis of an Essential Gene: a Majority of the Neisseria gonorrhoeae Major Outer Membrane Porin (PorB) Is Mutable
  13. Structure-Function Studies of the Neisseria gonorrhoeae Major Outer Membrane Porin
  14. Neisseria gonorrhoeae Metalloprotease NGO1686 Is Required for Full Piliation, and Piliation Is Required for Resistance to H2O2- and Neutrophil-Mediated Killing
  15. Neisseria gonorrhoeae RecQ Helicase HRDC Domains Are Essential for Efficient Binding and Unwinding of the pilE Guanine Quartet Structure Required for Pilin Antigenic Variation