Current affiliation: Massachusetts General Hospital/Harvard Medical SchoolSubject: Life SciencesPrimary location: United States
Published in:Trends in MicrobiologyPublication date:2008-04-01
Published in:Molecular MicrobiologyPublication date:2009-01-01
Published in:BMC GenomicsPublication date:2010-01-01
Analysis of eukaryotic gene expression profiles during Shigella infection in the presence and absence of an apoptosis inducer.
Published in:Infection and ImmunityPublication date:2007-03-05
Published in:Infection and ImmunityPublication date:2009-10-05
Published in:Molecular MicrobiologyPublication date:2012-05-23
OspE1 and OspE2 enhance adherence to epithelial cells
Previous work has demonstrated that bile salts induce the ability of Shigella flexneri to adhere and invade epithelial cells. This work demonstrate...
Published in:Proceedings of the National Academy of SciencesPublication date:2011-07-18
Published in:PLoS ONEPublication date:2012-11-16
Shigella flexerni expresses enterotoxins to induce watery diarrhea during infection. This study validated the function of previously described toxi...
Published in:Clinical Microbiology ReviewsPublication date:2016-07-27
Bacterial pathogens that infect the gastrointestinal tract must overcome harsh environmental conditions in order to successfully establish infectio...
Published in:Gut MicrobesPublication date:2016-09-22
OspD3, also known as Shigella Enterotoxin 2 (ShET2), is a virulence factor in Shigella flexneri that has been shown to induce watery diarrhea and r...
Published in:Infection and ImmunityPublication date:2017-03-27
This work demonstrates how Shigella flexneri resist bile salts and analyzes the changes in gene expression following bile salts exposure