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  1. Associations of COVID-19 Hospitalizations, ICU Admissions, and Mortality with Black and White Race and Their Mediation by Air Pollution and Other Risk Factors in the Louisiana Industrial Corridor, March 2020–August 2021
  2. Environmentally persistent free radicals enhance SARS-CoV-2 replication in respiratory epithelium
  3. Genomic Basis for Individual Differences in Susceptibility to the Neurotoxic Effects of Diesel Exhaust
  4. Metal-Free Biomass-Derived Environmentally Persistent Free Radicals (Bio-EPFRs) from Lignin Pyrolysis
  5. Risk factors among Black and White COVID-19 patients from a Louisiana Hospital System, March, 2020 – August, 2021
  6. IL-1β Promotes Expansion of IL-33+ Lung Epithelial Stem Cells after Respiratory Syncytial Virus Infection during Infancy
  7. Environmental Impacts on COVID-19: Mechanisms of Increased Susceptibility
  8. Exposure to combustion derived particulate matter exacerbates influenza infection in neonatal mice by inhibiting IL22 production
  9. Association between particulate matter containing EPFRs and neutrophilic asthma through AhR and Th17
  10. Particulate matter exposure predicts residence in high-risk areas for community acquired pneumonia among hospitalized children
  11. Developmental Hazard of Environmentally Persistent Free Radicals and Protective Effect of TEMPOL in Zebrafish Model
  12. Respiratory Syncytial Virus Infections Induce Hypermetabolism in Pediatric Upper Airways
  13. Altered gut microbiota in infants is associated with respiratory syncytial virus disease severity
  14. Altered Gut Microbiota in Infants is Associated with Respiratory Syncytial Virus Disease Severity
  15. Altered Gut Microbiota in Infants is Associated with Respiratory Syncytial Virus Disease Severity
  16. Respiratory Syncytial Virus Disease Severity Is Associated with Distinct CD8+ T-Cell Profiles
  17. Altered Gut Microbiota in Infants is Associated with Respiratory Syncytial Virus Disease Severity
  18. Elevated Levels of Type 2 Respiratory Innate Lymphoid Cells in Human Infants with Severe Respiratory Syncytial Virus Bronchiolitis
  19. Environmentally Persistent Free Radicals: Linking Air Pollution and Poor Respiratory Health?
  20. Corrigendum
  21. Particulate matter containing environmentally persistent free radicals induces AhR-dependent cytokine and reactive oxygen species production in human bronchial epithelial cells
  22. New mouse model of pulmonary hypertension induced by respiratory syncytial virus bronchiolitis
  23. Type I Interferon Potentiates IgA Immunity to Respiratory Syncytial Virus Infection During Infancy
  24. Future Research Directions in Pneumonia. NHLBI Working Group Report
  25. Radical containing combustion derived particulate matter enhance pulmonary Th17 inflammation via the aryl hydrocarbon receptor
  26. NKG2D Regulation of Lung Pathology and Dendritic Cell Function Following Respiratory Syncytial Virus Infection
  27. Environmentally Persistent Free Radicals: Insights on a New Class of Pollutants
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  29. A Scalable Field Study Protocol and Rationale for Passive Ambient Air Sampling: A Spatial Phytosampling for Leaf Data Collection
  30. Prolonged viral replication and longitudinal viral dynamic differences among respiratory syncytial virus infected infants
  31. Androgen Receptor Agonists Increase Lean Mass, Improve Cardiopulmonary Functions, and Extend Survival in Preclinical Models of Duchenne Muscular Dystrophy
  32. IL-4Rα on dendritic cells in neonates and Th2 immunopathology in respiratory syncytial virus infection
  33. Regulatory T cells and IL10 suppress pulmonary host defense during early-life exposure to radical containing combustion derived ultrafine particulate matter
  34. A Neonatal Murine Model of MRSA Pneumonia
  35. Spatiotemporal patterns of childhood asthma hospitalization and utilization in Memphis Metropolitan Area from 2005 to 2015
  36. Addressing Emerging Risks: Scientific and Regulatory Challenges Associated with Environmentally Persistent Free Radicals
  37. Exposure to environmentally persistent free radicals during gestation lowers energy expenditure and impairs skeletal muscle mitochondrial function in adult mice
  38. Plasma Biomarker Analysis in Pediatric ARDS: Generating Future Framework from a Pilot Randomized Control Trial of Methylprednisolone: A Framework for Identifying Plasma Biomarkers Related to Clinical Outcomes in Pediatric ARDS
  39. 14th congress of combustion by-products and their health effects—origin, fate, and health effects of combustion-related air pollutants in the coming era of bio-based energy sources
  40. Regulation of inflammatory biomarkers by intravenous methylprednisolone in pediatric ARDS patients: Results from a double-blind, placebo-controlled randomized pilot trial
  41. Using an External Exposome Framework to Examine Pregnancy-Related Morbidities and Mortalities: Implications for Health Disparities Research
  42. Autophagy is essential for ultrafine particle-induced inflammation and mucus hyperproduction in airway epithelium
  43. Respiratory Syncytial Virus Disease Is Mediated by Age-Variable IL-33
  44. Crawling with Virus: Translational Insights from a Neonatal Mouse Model on the Pathogenesis of Respiratory Syncytial Virus in Infants
  45. Building a better neonatal mouse model to understand infant respiratory syncytial virus disease
  46. Exposure to Deepwater Horizon Crude Oil Burnoff Particulate Matter Induces Pulmonary Inflammation and Alters Adaptive Immune Response
  47. Acute Lung Injury Results from Innate Sensing of Viruses by an ER Stress Pathway
  48. Exposure to Electronic Cigarettes Impairs Pulmonary Anti-Bacterial and Anti-Viral Defenses in a Mouse Model
  49. Serotonin 5-HT2 receptor activation prevents allergic asthma in a mouse model
  50. Deficiency of the Two-Pore-Domain Potassium Channel TREK-1 Promotes Hyperoxia-Induced Lung Injury
  51. Exposure to combustion generated environmentally persistent free radicals enhances severity of influenza virus infection
  52. Impaired gamma delta T cell-derived IL-17A and inflammasome activation during early respiratory syncytial virus infection in infants
  53. Limited Type I Interferons and Plasmacytoid Dendritic Cells during Neonatal Respiratory Syncytial Virus Infection Permit Immunopathogenesis upon Reinfection
  54. Combustion By-Products and Their Health Effects—Combustion Engineering and Global Health in the 21st Century
  55. Model Combustion-Generated Particulate Matter Containing Persistent Free Radicals Redox Cycle to Produce Reactive Oxygen Species
  56. Early-life exposure to combustion-derived particulate matter causes pulmonary immunosuppression
  57. Chronic Alcohol Induces M2 Polarization Enhancing Pulmonary Disease Caused by Exposure to Particulate Air Pollution
  58. IL-4Rα on CD4+ T cells plays a pathogenic role in respiratory syncytial virus reinfection in mice infected initially as neonates
  59. Radical-Containing Ultrafine Particulate Matter Initiates Epithelial-to-Mesenchymal Transitions in Airway Epithelial Cells
  60. Maternal exposure to combustion generated PM inhibits pulmonary Th1 maturation and concomitantly enhances postnatal asthma development in offspring
  61. Particulate Matter Containing Environmentally Persistent Free Radicals and Adverse Infant Respiratory Health Effects: A Review
  62. Model Particulate Matter Containing Persistent Free Radicals Exhibit Reactive Oxygen Species Production
  63. Innate IL-13 in virus-induced asthma?
  64. Environmentally persistent free radicals decrease cardiac function before and after ischemia/reperfusion injuryin vivo
  65. Environmentally persistent free radicals induce airway hyperresponsiveness in neonatal rat lungs
  66. The use of a neonatal mouse model to study respiratory syncytial virus infections
  67. Immunomodulation with IL-4Rα Antisense Oligonucleotide Prevents Respiratory Syncytial Virus-Mediated Pulmonary Disease
  68. In vitro and in vivo assessment of pulmonary risk associated with exposure to combustion generated fine particles
  69. Copper oxide nanoparticles induce oxidative stress and cytotoxicity in airway epithelial cells
  70. Modulation of lung inflammation by vessel dilator in a mouse model of allergic asthma
  71. Environmentally persistent free radicals amplify ultrafine particle mediated cellular oxidative stress and cytotoxicity
  72. Inchoate CD8+ T Cell Responses in Neonatal Mice Permit Influenza-Induced Persistent Pulmonary Dysfunction
  73. Post-allergen challenge inhibition of poly(ADP-ribose) polymerase harbors therapeutic potential for treatment of allergic airway inflammation
  74. Exposure of neonates to Respiratory Syncytial Virus is critical in determining subsequent airway response in adults
  75. Origin and Health Impacts of Emissions of Toxic By-Products and Fine Particles from Combustion and Thermal Treatment of Hazardous Wastes and Materials
  76. Pivotal Advance: Eosinophil infiltration of solid tumors is an early and persistent inflammatory host response
  77. Defining a Link with Asthma in Mice Congenitally Deficient in Eosinophils
  78. Hepatitis E Antibodies in Laboratory Rabbits from 2 US Vendors
  79. Methacholine-induced airway hyperresponsiveness is dependent on Gαq signaling
  80. A Causative Relationship Exists Between Eosinophils and the Development of Allergic Pulmonary Pathologies in the Mouse
  81. TH2-Mediated Pulmonary Inflammation Leads to the Differential Expression of Ribonuclease Genes by Alveolar Macrophages
  82. Th2 mediated pulmonary inflammation induces the differential expression of a unique eosinophil-associated ribonuclease gene
  83. Extensive Eosinophil Degranulation and Peroxidase-Mediated Oxidation of Airway Proteins Do Not Occur in a Mouse Ovalbumin-Challenge Model of Pulmonary Inflammation
  84. Mouse eosinophil-associated ribonucleases: a unique subfamily expressed during hematopoiesis
  85. Eosinophil Major Basic Protein-1 Does Not Contribute to Allergen-Induced Airway Pathologies in Mouse Models of Asthma
  86. Adrenomedullin Gene Expression Is Developmentally Regulated and Induced by Hypoxia in Rat Ventricular Cardiac Myocytes