All Stories

  1. GlycoDiveR: a modular R framework to analyze and visualize highly dimensional glycoproteomics data
  2. Understanding m / z Range Settings for MS/MS Scans: A Case Study with Intact Glycopeptides
  3. Profiling Glycoproteins Enriched by Multinanoparticle Protein Corona
  4. The protease Cathepsin K can debulk the cancer glycocalyx
  5. Extracting Informative Glycan-Specific Ions From Glycopeptide MS/MS Spectra With GlyCounter
  6. Sialoglycans on human T cells attenuate death programs executed through the Fas pathway
  7. Antibodies disrupt bacterial adhesion by ligand mimicry and allosteric interference
  8. Profiling glycoproteins enriched by multi-nanoparticle protein corona
  9. Understanding m/z range settings for MS/MS scans: a case study with intact glycopeptides
  10. Profiling glycoproteins enriched by surface-functionalized nanoparticles
  11. Sialoglycans on human T cells attenuate death programs executed through the Fas pathway
  12. Accelerating the stride toward functional glycoproteomics
  13. Improvements in Glycoproteomics through Architecture Changes to the Orbitrap Tribrid MS Platform
  14. Publisher Correction: irCLIP-RNP and Re-CLIP reveal patterns of dynamic protein assemblies on RNA
  15. Understanding m/z range settings for MS/MS scans: a case study with intact glycopeptides
  16. irCLIP-RNP and Re-CLIP reveal patterns of dynamic protein assemblies on RNA
  17. Extracting informative glycan-specific ions from glycopeptide MS/MS spectra with GlyCounter
  18. Comparative analysis of glycoproteomic software using a tailored glycan database
  19. Revisiting the Effect of Trypsin Digestion Buffers on Artificial Deamidation
  20. Antibodies disrupt bacterial adhesion by ligand mimicry and allosteric interference
  21. Autonomous Dissociation-type Selection for Glycoproteomics Using a Real-Time Library Search
  22. Sialylated glycoproteins suppress immune cell killing by binding to Siglec-7 and Siglec-9 in prostate cancer
  23. “Comparative Analysis of Glycoproteomic Software Using a Tailored Glycan Database”
  24. Instrumentation at the Leading Edge of Proteomics
  25. Instrumentation at the leading edge of proteomics
  26. Instrumentation at the leading edge of proteomics
  27. Microglia Mediate Contact-Independent Neuronal Network Remodeling via Secreted Neuraminidase-3 Associated with Extracellular Vesicles
  28. Elucidating the cellular determinants of targeted membrane protein degradation by lysosome-targeting chimeras
  29. Galectin-3 does not interact with RNA directly
  30. Microglia mediate contact-independent neuronal pruning via secreted Neuraminidase-3 associated with extracellular vesicles
  31. Design of a mucin-selective protease for targeted degradation of cancer-associated mucins
  32. Organism-wide, cell-type-specific secretome mapping of exercise training in mice
  33. The microenvironment dictates glycocalyx construction and immune surveillance
  34. Mutational screens highlight glycosylation as a modulator of colony-stimulating factor 3 receptor (CSF3R) activity
  35. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  36. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  37. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  38. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  39. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  40. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  41. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  42. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  43. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  44. Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  45. Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  46. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  47. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  48. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  49. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  50. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  51. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  52. Supplementary Data from Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  53. MYC-driven synthesis of Siglec ligands is a glycoimmune checkpoint
  54. The 2022 Nobel Prize in Chemistry—sweet!
  55. Measuring the multifaceted roles of mucin-domain glycoproteins in cancer
  56. Organism-wide secretome mapping uncovers pathways of tissue crosstalk in exercise
  57. Deciphering O-glycoprotease substrate preferences with O-Pair Search
  58. Antibody-lectin chimeras for glyco-immune checkpoint blockade
  59. The human disease gene LYSET is essential for lysosomal enzyme transport and viral infection
  60. Lysosomal cathepsin D mediates endogenous mucin glycodomain catabolism in mammals
  61. Deciphering O-glycoprotease substrate preferences with O-Pair Search
  62. Glycoproteomics
  63. Revealing the human mucinome
  64. Targeting hypersialylation in multiple myeloma represents a novel approach to enhance NK cell–mediated tumor responses
  65. Design of a mucin-selective protease for targeted degradation of cancer-associated mucins
  66. Structure-guided mutagenesis of a mucin-selective metalloprotease from Akkermansia muciniphila alters substrate preferences
  67. Deciphering O-glycoprotease substrate preferences with O-Pair Search
  68. Practical Effects of Intramolecular Hydrogen Rearrangement in Electron Transfer Dissociation-Based Proteomics
  69. Multiomics Analysis of Spatially Distinct Stromal Cells Reveals Tumor-Induced O-Glycosylation of the CDK4–pRB Axis in Fibroblasts at the Invasive Tumor Edge
  70. Protocol for cell type-specific labeling, enrichment, and proteomic profiling of plasma proteins in mice
  71. The CD22-IGF2R interaction is a therapeutic target for microglial lysosome dysfunction in Niemann-Pick type C
  72. LYTACs that engage the asialoglycoprotein receptor for targeted protein degradation
  73. Synthetic Siglec-9 Agonists Inhibit Neutrophil Activation Associated with COVID-19
  74. Revealing the human mucinome
  75. Genome-wide CRISPR screens reveal a specific ligand for the glycan-binding immune checkpoint receptor Siglec-7
  76. Modulation of immune cell reactivity with cis -binding Siglec agonists
  77. A Pragmatic Guide to Enrichment Strategies for Mass Spectrometry–Based Glycoproteomics
  78. Synthetic Siglec-9 Agonists Inhibit Neutrophil Activation Associated with COVID-19
  79. Cell type-selective secretome profiling in vivo
  80. Electron-Based Dissociation Is Needed for O-Glycopeptides Derived from OpeRATOR Proteolysis
  81. O-Pair Search with MetaMorpheus for O-glycopeptide characterization
  82. Cell type-selective secretome profiling in vivo
  83. Optical Fiber-Enabled Photoactivation of Peptides and Proteins
  84. Lysosome Targeting Chimeras (LYTACs) That Engage a Liver-Specific Asialoglycoprotein Receptor for Targeted Protein Degradation
  85. Lysosome-targeting chimaeras for degradation of extracellular proteins
  86. Electron-Based Dissociation Is Needed for O-Glycopeptides Derived from OpeRATOR Proteolysis
  87. Top-Down Characterization of an Intact Monoclonal Antibody Using Activated Ion Electron Transfer Dissociation
  88. Optimal Dissociation Methods Differ for N- and O-Glycopeptides
  89. Optimal Dissociation Methods Differ for N- and O-glycopeptides
  90. O-Pair Search with MetaMorpheus for O-glycopeptide Characterization
  91. Optimal Dissociation Methods Differ for N- and O-glycopeptides
  92. Broad and thematic remodeling of the surfaceome and glycoproteome on isogenic cells transformed with driving proliferative oncogenes
  93. Lysosome Targeting Chimeras (LYTACs) for the Degradation of Secreted and Membrane Proteins
  94. Broad and thematic remodeling of the surface glycoproteome on isogenic cells transformed with driving proliferative oncogenes
  95. Mutating stress-defense regulators in yeast improves biofuels engineering
  96. Interactive Peptide Spectral Annotator: A Versatile Web-Based Tool for Proteomic Applications
  97. Lysosome Targeting Chimeras (LYTACs) for the Degradation of Secreted and Membrane Proteins
  98. Lysosome Targeting Chimeras (LYTACs) for the Degradation of Secreted and Membrane Proteins
  99. Capturing site-specific heterogeneity with large-scale N-glycoproteome analysis
  100. Rewired cellular signaling coordinates sugar and hypoxic responses for anaerobic xylose fermentation in yeast
  101. Top-Down Characterization of Proteins with Intact Disulfide Bonds Using Activated-Ion Electron Transfer Dissociation
  102. The Value of Activated Ion Electron Transfer Dissociation for High-Throughput Top-Down Characterization of Intact Proteins
  103. Improved Precursor Characterization for Data-Dependent Mass Spectrometry
  104. The Role of Electron Transfer Dissociation in Modern Proteomics
  105. Sequencing Larger Intact Proteins (30-70 kDa) with Activated Ion Electron Transfer Dissociation
  106. Activated Ion-Electron Transfer Dissociation Enables Comprehensive Top-Down Protein Fragmentation
  107. Negative Electron Transfer Dissociation Sequencing of Increasingly Sulfated Glycosaminoglycan Oligosaccharides on an Orbitrap Mass Spectrometer
  108. Implementation of Activated Ion Electron Transfer Dissociation on a Quadrupole-Orbitrap-Linear Ion Trap Hybrid Mass Spectrometer
  109. Phosphoproteomics with Activated Ion Electron Transfer Dissociation
  110. Sulfur Pentafluoride is a Preferred Reagent Cation for Negative Electron Transfer Dissociation
  111. Multi-omics Evidence for Inheritance of Energy Pathways in Red Blood Cells
  112. Full-Featured Search Algorithm for Negative Electron-Transfer Dissociation
  113. Genome Sequence and Analysis of a Stress-Tolerant, Wild-Derived Strain of Saccharomyces cerevisiae Used in Biofuels Research
  114. Proteomics Moves into the Fast Lane
  115. Mitochondrial protein hyperacetylation in the failing heart
  116. Enhanced Dissociation of Intact Proteins with High Capacity Electron Transfer Dissociation
  117. Phosphoproteomics in the Age of Rapid and Deep Proteome Profiling
  118. The Negative Mode Proteome with Activated Ion Negative Electron Transfer Dissociation (AI-NETD)
  119. A Calibration Routine for Efficient ETD in Large-Scale Proteomics
  120. Activated Ion Electron Transfer Dissociation for Improved Fragmentation of Intact Proteins
  121. Coupling Capillary Zone Electrophoresis with Electron Transfer Dissociation and Activated Ion Electron Transfer Dissociation for Top-Down Proteomics
  122. Neutron-Encoded Mass Signatures for Quantitative Top-Down Proteomics