All Stories

  1. Sensory neurons regulate stimulus-dependent humoral immunity in mouse models of bacterial infection and asthma
  2. Non-canonical IL-22 receptor signaling remodels the mucosal barrier during fungal immunosurveillance
  3. Synergistic cross-kingdom host cell damage betweenCandida albicansandEnterococcus faecalis
  4. The Role of Interferon-γ in Autoimmune Polyendocrine Syndrome Type 1
  5. Sensory neurons regulate stimulus-dependent humoral immunity
  6. Candida albicans stimulates formation of a multi-receptor complex that mediates epithelial cell invasion during oropharyngeal infection
  7. Candida albicansstimulates the formation of a multi-receptor complex that mediates epithelial cell invasion during oropharyngeal infection
  8. IL-23 signaling prevents ferroptosis-driven renal immunopathology during candidiasis
  9. Immunosurveillance of Candida albicans commensalism by the adaptive immune system
  10. Serum bridging molecules drive candidal invasion of human but not mouse endothelial cells
  11. Editorial: Immunological Memory to Fungal Infections and Vaccine Development
  12. Control of β-glucan exposure by the endo-1,3-glucanase Eng1 in Candida albicans modulates virulence
  13. IL-23 signaling prevents ferroptosis-driven renal immunopathology during candidiasis
  14. The Globular C1q Receptor Is Required for Epidermal Growth Factor Receptor Signaling during Candida albicans Infection
  15. Identification of Serum Bridging Molecules that Mediate Human Endothelial Cell Invasion byCandidaspecies
  16. Response to Comments on “Aberrant type 1 immunity drives susceptibility to mucosal fungal infections”
  17. Rapid proliferation due to better metabolic adaptation results in full virulence of a filament-deficient Candida albicans strain
  18. The globular C1q receptor is required for epidermal growth factor receptor signaling during Candida albicans infection
  19. Activation of EphA2-EGFR signaling in oral epithelial cells by Candida albicans virulence factors
  20. Mucoricin is a ricin-like toxin that is critical for the pathogenesis of mucormycosis
  21. Aberrant type 1 immunity drives susceptibility to mucosal fungal infections
  22. Mucoricin is a Ricin-Like Toxin that is Critical for the Pathogenesis of Mucormycosis
  23. Mucosal IgA Prevents Commensal Candida albicans Dysbiosis in the Oral Cavity
  24. Interleukin-26 activates macrophages and facilitates killing of Mycobacterium tuberculosis
  25. Control of β-glucan exposure by the endo-1,3-glucanase Eng1 inCandida albicansmodulates virulence
  26. GRP78 and Integrins Play Different Roles in Host Cell Invasion during Mucormycosis
  27. Candidalysin Is Required for Neutrophil Recruitment and Virulence During Systemic Candida albicans Infection
  28. EphA2 Is a Neutrophil Receptor for Candida albicans that Stimulates Antifungal Activity during Oropharyngeal Infection
  29. Selection of Candida albicans trisomy during oropharyngeal infection results in a commensal-like phenotype
  30. Candida albicans at Host Barrier Sites: Pattern Recognition Receptors and Beyond
  31. Selection of Candida albicans Trisomy during Oropharyngeal Infection Results in a Commensal-Like Phenotype
  32. Inhibition of EGFR Signaling Protects from Mucormycosis
  33. Author Correction: EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  34. The Hyr1 protein from the fungus Candida albicans is a cross kingdom immunotherapeutic target for Acinetobacter bacterial infection
  35. Candida albicans White-Opaque Switching Influences Virulence but Not Mating during Oropharyngeal Candidiasis
  36. Publisher Correction: EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  37. EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  38. Innate Immunity to Mucosal Candida Infections
  39. The Aryl Hydrocarbon Receptor Governs Epithelial Cell Invasion during Oropharyngeal Candidiasis
  40. Oropharyngeal Candidiasis: Fungal Invasion and Epithelial Cell Responses
  41. AMPlified Defense: Antimicrobial Peptides During Candida albicans Infection
  42. Aspergillus fumigatus CalA binds to integrin α5β1 and mediates host cell invasion
  43. Candida albicans responds to glycostructure damage by Ace2‐mediated feedback regulation of Cek1 signaling
  44. Domains of CaMsb2
  45. Candida and AMPs
  46. C. albicans Mucin Msb2
  47. Msb2 Shedding