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  1. Sympathetic neurons control adaptive immunity in response to Streptococcus pneumoniae infection by regulating T cell and B cell effector function
  2. Sensory neurons regulate stimulus-dependent humoral immunity in mouse models of bacterial infection and asthma
  3. Non-canonical IL-22 receptor signaling remodels the mucosal barrier during fungal immunosurveillance
  4. Synergistic cross-kingdom host cell damage betweenCandida albicansandEnterococcus faecalis
  5. The Role of Interferon-γ in Autoimmune Polyendocrine Syndrome Type 1
  6. Sensory neurons regulate stimulus-dependent humoral immunity
  7. Candida albicans stimulates formation of a multi-receptor complex that mediates epithelial cell invasion during oropharyngeal infection
  8. Candida albicansstimulates the formation of a multi-receptor complex that mediates epithelial cell invasion during oropharyngeal infection
  9. IL-23 signaling prevents ferroptosis-driven renal immunopathology during candidiasis
  10. Immunosurveillance of Candida albicans commensalism by the adaptive immune system
  11. Serum bridging molecules drive candidal invasion of human but not mouse endothelial cells
  12. Editorial: Immunological Memory to Fungal Infections and Vaccine Development
  13. Control of β-glucan exposure by the endo-1,3-glucanase Eng1 in Candida albicans modulates virulence
  14. IL-23 signaling prevents ferroptosis-driven renal immunopathology during candidiasis
  15. The Globular C1q Receptor Is Required for Epidermal Growth Factor Receptor Signaling during Candida albicans Infection
  16. Identification of Serum Bridging Molecules that Mediate Human Endothelial Cell Invasion byCandidaspecies
  17. Response to Comments on “Aberrant type 1 immunity drives susceptibility to mucosal fungal infections”
  18. Rapid proliferation due to better metabolic adaptation results in full virulence of a filament-deficient Candida albicans strain
  19. The globular C1q receptor is required for epidermal growth factor receptor signaling during Candida albicans infection
  20. Activation of EphA2-EGFR signaling in oral epithelial cells by Candida albicans virulence factors
  21. Mucoricin is a ricin-like toxin that is critical for the pathogenesis of mucormycosis
  22. Aberrant type 1 immunity drives susceptibility to mucosal fungal infections
  23. Mucoricin is a Ricin-Like Toxin that is Critical for the Pathogenesis of Mucormycosis
  24. Mucosal IgA Prevents Commensal Candida albicans Dysbiosis in the Oral Cavity
  25. Interleukin-26 activates macrophages and facilitates killing of Mycobacterium tuberculosis
  26. Control of β-glucan exposure by the endo-1,3-glucanase Eng1 inCandida albicansmodulates virulence
  27. GRP78 and Integrins Play Different Roles in Host Cell Invasion during Mucormycosis
  28. Candidalysin Is Required for Neutrophil Recruitment and Virulence During Systemic Candida albicans Infection
  29. EphA2 Is a Neutrophil Receptor for Candida albicans that Stimulates Antifungal Activity during Oropharyngeal Infection
  30. Selection of Candida albicans trisomy during oropharyngeal infection results in a commensal-like phenotype
  31. Candida albicans at Host Barrier Sites: Pattern Recognition Receptors and Beyond
  32. Selection of Candida albicans Trisomy during Oropharyngeal Infection Results in a Commensal-Like Phenotype
  33. Inhibition of EGFR Signaling Protects from Mucormycosis
  34. Author Correction: EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  35. The Hyr1 protein from the fungus Candida albicans is a cross kingdom immunotherapeutic target for Acinetobacter bacterial infection
  36. Candida albicans White-Opaque Switching Influences Virulence but Not Mating during Oropharyngeal Candidiasis
  37. Publisher Correction: EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  38. EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  39. Innate Immunity to Mucosal Candida Infections
  40. The Aryl Hydrocarbon Receptor Governs Epithelial Cell Invasion during Oropharyngeal Candidiasis
  41. Oropharyngeal Candidiasis: Fungal Invasion and Epithelial Cell Responses
  42. AMPlified Defense: Antimicrobial Peptides During Candida albicans Infection
  43. Aspergillus fumigatus CalA binds to integrin α5β1 and mediates host cell invasion
  44. Candida albicans responds to glycostructure damage by Ace2‐mediated feedback regulation of Cek1 signaling
  45. Domains of CaMsb2
  46. Candida and AMPs
  47. C. albicans Mucin Msb2
  48. Msb2 Shedding