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  1. Autophagy modulates lipid metabolism to maintain metabolic flexibility for Lkb1-deficient Kras-driven lung tumorigenesis
  2. Amino acid metabolites, mTORC1 and aging
  3. The amino acid metabolite homocysteine activates mTORC1 to inhibit autophagy and form abnormal proteins in human neurons and mice
  4. Dysregulation of Nutrient Sensing and CLEARance in Presenilin Deficiency
  5. Cellular metabolism and lysosomal mTOR signaling