All Stories

  1. Metabolic Mitigation of Staphylococcus aureus Vancomycin Intermediate-Level Susceptibility
  2. Staphylococcus aureus Metabolic Adaptations during the Transition from a Daptomycin Susceptibility Phenotype to a Daptomycin Nonsusceptibility Phenotype
  3. The Catabolite Control Protein E (CcpE) Affects Virulence Determinant Production and Pathogenesis of Staphylococcus aureus
  4. A Dysfunctional Tricarboxylic Acid Cycle Enhances Fitness of Staphylococcus epidermidis during β-Lactam Stress
  5. Influence of Iron and Aeration on Staphylococcus aureus Growth, Metabolism, and Transcription
  6. Identification of Low‐Molecular‐Weight Compounds Inhibiting Growth of Corynebacteria: Potential Lead Compounds for Antibiotics
  7. Catabolite Control Protein E (CcpE) Is a LysR-type Transcriptional Regulator of Tricarboxylic Acid Cycle Activity in Staphylococcus aureus
  8. Growth and Preparation of Staphylococcus epidermidis for NMR Metabolomic Analysis
  9. A Dysfunctional Tricarboxylic Acid Cycle Enhances Fitness of Staphylococcus epidermidis During β-Lactam Stress
  10. Reductive evolution and the loss of PDC/PAS domains from the genus Staphylococcus
  11. Cultivation conditions and the diffusion of oxygen into culture media: The rationale for the flask-to-medium ratio in microbiology
  12. CcpA coordinates central metabolism and biofilm formation in Staphylococcus epidermidis
  13. Staphylococcal response to oxidative stress
  14. CcpA coordinates central metabolism and biofilm formation in Staphylococcus epidermidis
  15. RpiR Homologues May Link Staphylococcus aureus RNAIII Synthesis and Pentose Phosphate Pathway Regulation
  16. NMR Analysis of a Stress Response Metabolic Signaling Network
  17. TCA cycle inactivation in Staphylococcus aureus alters nitric oxide production in RAW 264.7 cells
  18. Using NMR Metabolomics to Investigate Tricarboxylic Acid Cycle-dependent Signal Transduction in Staphylococcus epidermidis
  19. Direct Targets of CodY in Staphylococcus aureus
  20. Direct Targets of CodY in Staphylococcus aureus
  21. Tricarboxylic Acid Cycle-Dependent Synthesis of Staphylococcus aureus Type 5 and 8 Capsular Polysaccharides
  22. The Biology of Staphylococci
  23. Tricarboxylic Acid Cycle-Dependent Attenuation of Staphylococcus aureus In Vivo Virulence by Selective Inhibition of Amino Acid Transport
  24. Staphylococcus aureus ClpC ATPase is a late growth phase effector of metabolism and persistence
  25. Tricarboxylic Acid Cycle-Dependent Regulation of Staphylococcus epidermidis Polysaccharide Intercellular Adhesin Synthesis
  26. Staphylococcus aureus CodY Negatively Regulates Virulence Gene Expression
  27. Staphylococcus epidermidis saeR Is an Effector of Anaerobic Growth and a Mediator of Acute Inflammation
  28. Staphylococcus aureus Biofilm Metabolism and the Influence of Arginine on Polysaccharide Intercellular Adhesin Synthesis, Biofilm Formation, and Pathogenesis
  29. Pathological Processes Related to Redox
  30. Vancomycin-Intermediate Staphylococcus aureus Strains Have Impaired Acetate Catabolism: Implications for Polysaccharide Intercellular Adhesin Synthesis and Autolysis
  31. Very Low Ethanol Concentrations Affect the Viability and Growth Recovery in Post-Stationary-Phase Staphylococcus aureus Populations
  32. Staphylococcus aureus ClpC Is Required for Stress Resistance, Aconitase Activity, Growth Recovery, and Death
  33. Staphylococcus epidermidis Polysaccharide Intercellular Adhesin Production Significantly Increases during Tricarboxylic Acid Cycle Stress
  34. Progress toward Characterization of the Group A Streptococcus Metagenome: Complete Genome Sequence of a Macrolide‐Resistant Serotype M6 Strain
  35. Synthesis and Deformylation of Staphylococcus aureus δ-Toxin Are Linked to Tricarboxylic Acid Cycle Activity
  36. Quorum‐Sensing Control of Biofilm Factors in Staphylococcus epidermidis
  37. Correlation of Acetate Catabolism and Growth Yield in Staphylococcus aureus: Implications for Host-Pathogen Interactions
  38. Staphylococcus aureus Aconitase Inactivation Unexpectedly Inhibits Post-Exponential-Phase Growth and Enhances Stationary-Phase Survival
  39. In Vitro Serial Passage of Staphylococcus aureus: Changes in Physiology, Virulence Factor Production, and agr Nucleotide Sequence