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  1. Natural Killer Cells Are Dispensable for Virus Control in Rag2−/− Mice During Primary RSV Infection
  2. Sustained Lung Inflammation Post‐SARS‐CoV‐2 Infection in Mice Is Associated with Increased Pulmonary T Cells
  3. RORing for oral tolerance
  4. Type I interferons induced upon respiratory viral infection impair lung metastatic initiation
  5. SARS-CoV-2 strains bearing Omicron BA.1 spike replicate in C57BL/6 mice
  6. TLR7 Promotes Emergency Myelopoiesis and Antiviral Immunity
  7. Formulation, inflammation, and RNA sensing impact the immunogenicity of self-amplifying RNA vaccines
  8. Levels of Influenza A Virus Defective Viral Genomes Determine Pathogenesis in the BALB/c Mouse Model
  9. Type I interferon receptor signalling deficiency results in dysregulated innate immune responses to SARS‐CoV‐2 in mice
  10. The Use of Precision‐Cut Lung Slices for Studying Innate Immunity to Viral Infections
  11. Type I interferons and MAVS signaling are necessary for tissue resident memory CD8+ T cell responses to RSV infection
  12. Lentiviral and AAV-mediated expression of palivizumab offer protection against Respiratory Syncytial Virus infection
  13. Neutrophils in respiratory viral infections
  14. Rapidly Deployable Mouse Models of SARS-CoV-2 Infection Add Flexibility to the COVID-19 Toolbox
  15. R848 or influenza virus can induce potent innate immune responses in the lungs of neonatal mice
  16. Neutrophilic inflammation in the respiratory mucosa predisposes to RSV infection
  17. MAVS Deficiency Is Associated With a Reduced T Cell Response Upon Secondary RSV Infection in Mice
  18. Neutrophils do not impact viral load or the peak of disease severity during RSV infection
  19. Chemokine regulation of inflammation during respiratory syncytial virus infection
  20. Neutrophil recruitment and activation are differentially dependent on MyD88/TRIF and MAVS signaling during RSV infection
  21. Internal genes of a highly pathogenic H5N1 influenza virus determine high viral replication in myeloid cells and severe outcome of infection in mice
  22. Type I interferon is required for T helper (Th) 2 induction by dendritic cells
  23. Protective and Harmful Immunity to RSV Infection
  24. Type I Interferons as Regulators of Lung Inflammation
  25. Respiratory syncytial virus infection: an innate perspective
  26. Alveolar Macrophages Can Control Respiratory Syncytial Virus Infection in the Absence of Type I Interferons
  27. T cell responses are elicited against Respiratory Syncytial Virus in the absence of signalling through TLRs, RLRs and IL-1R/IL-18R
  28. Alveolar macrophage–derived type I interferons orchestrate innate immunity to RSV through recruitment of antiviral monocytes
  29. DNGR-1 is dispensable for CD8+T-cell priming during respiratory syncytial virus infection
  30. Alpha/Beta Interferon Receptor Signaling Amplifies Early Proinflammatory Cytokine Production in the Lung during Respiratory Syncytial Virus Infection
  31. Regulatory T Cells Prevent Th2 Immune Responses and Pulmonary Eosinophilia during Respiratory Syncytial Virus Infection in Mice
  32. Neonatal antibody responses are attenuated by interferon-γ produced by NK and T cells during RSV infection
  33. Defective immunoregulation in RSV vaccine-augmented viral lung disease restored by selective chemoattraction of regulatory T cells
  34. IL-10 Regulates Viral Lung Immunopathology during Acute Respiratory Syncytial Virus Infection in Mice
  35. Regulatory T cells expressing granzyme B play a critical role in controlling lung inflammation during acute viral infection
  36. CD25+ Natural Regulatory T Cells Are Critical in Limiting Innate and Adaptive Immunity and Resolving Disease following Respiratory Syncytial Virus Infection
  37. Type I interferons produced by hematopoietic cells protect mice against lethal infection by mammalian reovirus
  38. Phenotype and function of intestinal dendritic cells
  39. Liver Dendritic Cells Present Bacterial Antigens and Produce Cytokines upon Salmonella Encounter
  40. The response of natural killer T cells to glycolipid antigens is characterized by surface receptor down-modulation and expansion
  41. Dendritic cells as inducers of antimicrobial immunity in vivo
  42. Salmonella typhimurium -induced cytokine production and surface molecule expression by murine macrophages
  43. The influence of curli, a MHC-I-binding bacterial surface structure, on macrophage–T cell interactions
  44. Salmonella enterica Serovar Typhimurium-Induced Maturation of Bone Marrow-Derived Dendritic Cells
  45. Salmonella infection of bone marrow-derived macrophages and dendritic cells: influence on antigen presentation and initiating an immune response