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  1. REGULATION OF AMH BY OOCYTE SPECIFIC GROWTH FACTORS IN HUMAN PRIMARY CUMULUS CELLS
  2. IGF1R Expression in Ovarian Granulosa Cells Is Essential for Steroidogenesis, Follicle Survival, and Fertility in Female Mice
  3. Genome-wide interactions between FSH and insulin-like growth factors in the regulation of human granulosa cell differentiation
  4. GATA4 and GATA6 Knockdown During Luteinization Inhibits Progesterone Production and Gonadotropin Responsiveness in the Corpus Luteum of Female Mice1
  5. FSH Regulates IGF-2 Expression in Human Granulosa Cells in an AKT-Dependent Manner
  6. IGF1R Signaling Is Necessary for FSH-Induced Activation of AKT and Differentiation of Human Cumulus Granulosa Cells
  7. GATA4 and GATA6 Silencing in Ovarian Granulosa Cells Affects Levels of mRNAs Involved in Steroidogenesis, Extracellular Structure Organization, IGF-I Activity, and Apoptosis
  8. Testosterone-Dependent Interaction between Androgen Receptor and Aryl Hydrocarbon Receptor Induces Liver Receptor Homolog 1 Expression in Rat Granulosa Cells
  9. IGF-I Signaling Is Essential for FSH Stimulation of AKT and Steroidogenic Genes in Granulosa Cells
  10. Intracellular Crosstalk Between FSH and Androgens in Granulosa Cells.
  11. GATA-4 Silencing Affects Apoptosis, Steroidogenesis, and IGF-1 Signaling Pathways in Ovarian Granulosa Cells.
  12. IGF-1 Signaling Is Required for FSH Induction of Cyp19 Expression in Granulosa Cells.
  13. Loss of GATA-6 and GATA-4 in Granulosa Cells Blocks Folliculogenesis, Ovulation, and Follicle Stimulating Hormone Receptor Expression Leading to Female Infertility
  14. The Long and Short of the Prolactin Receptor: The Corpus Luteum Needs Them Both!1
  15. Tissue physiology and pathology of aromatase
  16. The Role of GATA-4 and GATA-6 in Granulosa Cell Function.
  17. Testosterone, not 5alpha-dihydrotestosterone Stimulates Liver Receptor homolog-1 Expression by Activating the Androgen Receptor in Primary Rat Granulosa Cells.
  18. Activation of the PI3K/Akt/mTOR Pathway by Insulin Is Crucial for Amplification of FSH-Induced Differentiation, but Not Proliferation, of Ovarian Granulosa Cells.
  19. Testosterone, not 5α-Dihydrotestosterone, Stimulates LRH-1 Leading to FSH-Independent Expression of Cyp19 and P450scc in Granulosa Cells
  20. Testosterone, not 5α-Dihydrotestosterone, Stimulates LRH-1 Leading to FSH-Independent Expression of Cyp19 and P450scc in Granulosa Cells
  21. Androgens Stimulate Aromatase and P450scc in Primary Rat Granulosa Cells Independently of Follicle Stimulating Hormone.
  22. Opposite Effects of Prolactin (Prl) on the Expression of Aromatase and Liver Receptor Homolog 1 (LRH-1) in Luteal Cells.
  23. Regulation of Transcription Factors and Repression of Sp1 by Prolactin Signaling Through the Short Isoform of Its Cognate Receptor
  24. Molecular Control of Corpus Luteum Function
  25. Hormonal and Molecular Regulation of the Cytochrome P450 Aromatase Gene Expression in the Ovary
  26. Prolactin signaling through the short isoform of the mouse prolactin receptor regulates DNA binding of specific transcription factors, often with opposite effects in different reproductive issues
  27. Aromatase expression in the ovary: Hormonal and molecular regulation
  28. Follicle-Stimulating Hormone-Induced Activation of Gata4 Contributes in the Up-Regulation of Cyp19 Expression in Rat Granulosa Cells
  29. The Molecular Control of Corpus Luteum Formation, Function, and Regression
  30. Prostaglandin E2 increases cyp19 expression in rat granulosa cells: Implication of GATA-4
  31. Expression and Regulation of Progestin Membrane Receptors in the Rat Corpus Luteum
  32. Cloning and Characterization of a 5′ Regulatory Region of the Prolactin Receptor-Associated Protein/17β Hydroxysteroid Dehydrogenase 7 Gene
  33. Changes in the Expression of Steroidogenic and Antioxidant Genes in the Mouse Corpus Luteum During Luteolysis1
  34. In Vivoandin VitroInhibition ofcyp19Gene Expression by Prostaglandin F2αin Murine Luteal Cells: Implication of GATA-4
  35. Prostaglandin F2α(PGF2α) and Prolactin Signaling: PGF2α-Mediated Inhibition of Prolactin Receptor Expression in the Corpus Luteum
  36. The Cyclin-dependent Kinase Inhibitors p27Kip1and p21Cip1Cooperate to Restrict Proliferative Life Span in Differentiating Ovarian Cells
  37. A Calcium/Calmodulin-dependent Activation of ERK1/2 Mediates JunD Phosphorylation and Induction ofnur77and20α-hsdGenes by Prostaglandin F2αin Ovarian Cells
  38. Luteal Expression of Cytochrome P450 Side-Chain Cleavage, Steroidogenic Acute Regulatory Protein, 3β-Hydroxysteroid Dehydrogenase, and 20α-Hydroxysteroid Dehydrogenase Genes in Late Pregnant Rats: Effect of Luteinizing Hormone and RU4861
  39. Opposite Effect of Prolactin and Prostaglandin F2αon the Expression of Luteal Genes as Revealed by Rat cDNA Expression Array
  40. Prostaglandin F2α-induced Expression of 20α-Hydroxysteroid Dehydrogenase Involves the Transcription Factor NUR77
  41. Progesterone receptor is not required for progesterone action in the rat corpus luteum of pregnancy☆
  42. Luteinizing Hormone Inhibits Conversion of Pregnenolone to Progesterone in Luteal Cells from Rats on Day 19 of Pregnancy1
  43. Antisteroidogenic Action of Nitric Oxide on Human Corpus Luteum In Vitro: Mechanism of Action
  44. Dual regulation of luteal progesterone production by androstenedione during spontaneous and RU486-induced luteolysis in pregnant rats
  45. Luteolytic action of RU486: Modulation of luteal 3β-hydroxysteroid dehydrogenase and 20α-hydroxysteroid dehydrogenase activities in late pregnant rats