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  1. Transcriptomic Approach in Understanding Fabry Nephropathy: A Review of the Literature and Proof-of-Concept
  2. Telomere Length, Oxidative Stress, and Kidney Damage Biomarkers in Fabry Nephropathy
  3. Dynamics of Leukocyte Telomere Length in Patients with Fabry Disease
  4. Urinary-derived extracellular vesicles reveal a distinct microRNA signature associated with the development and progression of Fabry nephropathy
  5. An expert consensus on practical clinical recommendations and guidance for patients with classic Fabry disease
  6. Renoprotective Effect of Agalsidase Alfa: A Long-Term Follow-Up of Patients with Fabry Disease
  7. Podocyturia in Fabry disease: a 10-year follow-up
  8. Urinary Extracellular Vesicles and Their miRNA Cargo in Patients with Fabry Nephropathy
  9. Characteristics of Vascular Phenotype in Fabry Patients
  10. Early indicators of disease progression in Fabry disease that may indicate the need for disease-specific treatment initiation: findings from the opinion-based PREDICT-FD modified Delphi consensus initiative
  11. Biomarkers of Fabry Nephropathy: Review and Future Perspective
  12. Cardiomyopathy and kidney function in agalsidase beta‐treated female Fabry patients: a pre‐treatment vs. post‐treatment analysis
  13. Switching from agalsidase alfa to pegunigalsidase alfa for treating Fabry disease: One year of treatment data from BRIDGE, a phase III open label study
  14. Aging in Fabry Disease: Role of Telomere Length, Telomerase Activity, and Kidney Disease
  15. Analysis of the baseline characteristics of Fabry disease patients screened for the pegunigalsidase alfa phase III BALANCE study
  16. Pegunigalsidase alfa for the treatment of Fabry disease: Preliminary results from a phase III open label, switch over study from agalsidase alfa
  17. Renal and cardiac outcomes in female patients with Fabry disease treated with agalsidase beta: A Fabry registry analysis of pre- versus post-treatment comparison
  18. Fabry disease: diagnostic methods in nephrology practice
  19. Paricalcitol as an Antiproteinuric Agent Can Result in the Deterioration of Renal and Heart Function in a Patient with Fabry Disease
  20. Renal Replacement Therapy in Slovenia: Excerpts From 2013 Data
  21. Anti-proteinuric therapy and Fabry nephropathy; factors associated with preserved kidney function during agalsidase-beta therapy
  22. Antiproteinuric therapy and Fabry nephropathy: factors associated with preserved kidney function during agalsidase-beta therapy
  23. Recommendation for initiation and cessation of enzyme replacement therapy
  24. Exocrine pancreatic insufficiency is not a cause of abdominal complaints in patients with Fabry disease
  25. Recommendations for initiation and cessation of enzyme replacement therapy in patients with Fabry disease: the European Fabry Working Group consensus document
  26. Exocrine pancreatic insufficiency is not a cause of abdominal complaints in patients with fabry disease
  27. Renal Replacement Therapy in Slovenia: Excerpts From 2011 Data
  28. Recommendations on Reintroduction of Agalsidase Beta for Patients with Fabry Disease in Europe, Following a Period of Shortage
  29. Could Iron Deficiency Also Affect Platelet-Related Hemostasis Impairment in Hemodialysis Patients?: Reply
  30. Renal outcomes of agalsidase beta treatment for Fabry disease: role of proteinuria and timing of treatment initiation
  31. Urinary Total Globotriaosylceramide and Isoforms to Identify Women With Fabry Disease: A Diagnostic Test Study
  32. Prognostic Indicators of Renal Disease Progression in Adults with Fabry Disease: Natural History Data from the Fabry Registry
  33. Association Between Platelet-Related Hemostasis Impairment and Functional Iron Deficiency in Hemodialysis Patients
  34. 143. End stage renal disease in patients with Fabry disease: Natural history data from the Fabry registry
  35. End-stage renal disease in patients with Fabry disease: natural history data from the Fabry Registry
  36. Scoring system for renal pathology in Fabry disease: report of the International Study Group of Fabry Nephropathy (ISGFN)
  37. A successful treatment of life-threatening bleeding from polycystic kidneys with antifibrinolytic agent tranexamic acid
  38. Epoetin Responsiveness in Peritoneal Dialysis Patients: A Multi-center Slovenian Study
  39. The effect of long term, low-dose tranexamic acid treatment on platelet dysfunction and haemoglobin levels in haemodialysis patients
  40. The study of anaemia-related haemostasis impairment in haemodialysis patients by in vitro closure time test
  41. Remarkable variability in renal disease in a large Slovenian family with Fabry disease
  42. Severe Diabetic Ketoacidosis Associated With Acute Myocardial Necrosis
  43. Tranexamic acid is beneficial as adjunctive therapy in treating major upper gastrointestinal bleeding in dialysis patients
  44. Treatment of subdural and intracerebral haematomas in a haemodialysis patient with tranexamic acid
  45. Successful treatment of bleeding from colonic angiodysplasias with tranexamic acid in a hemodialysis patient