What is it about?
Oxidative stress caused by BPA is may be the reason of delayed testis development Reproductive toxicity during prepubertal and pubertal period causes significant alterations in sexual maturation and fertility. Bisphenol A has been associated with decreased sperm production, damaged testicular cells, perturbed hormone levels and reduced fertility. Its role in sexual maturity and duration before first successful mating is still elusive. The present study aimed to investigate BPA induced alteration in testicular development and fertility at critical peroid . Histological observations indicated interferences at various stages during spermatogenesis along with presence of tubular vacuolization, indicative of cellular toxicity. Higher doses of BPA inflicted more damage, likewise, longer duration amplified severity of adverse activity. BPA administration at critical period of reproductive development in male rats can adversely affect testicular functions leading to delayed and reduced fertility.
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Why is it important?
Its important that we keep safe our children from bpa products we used for their natural development
Perspectives
My work explains the role of bisphenol A in delayed and reduced fertility and it may be a reason of reduced fertility in human population also.
Manish Sharma
University of Rajasthan
Read the Original
This page is a summary of: Role of Bisphenol A in Regulation of Arrival of Sexual Maturity and Fertility in Male Albino Rats, Journal of Pharmaceutical Research International, December 2021, Sciencedomain International,
DOI: 10.9734/jpri/2021/v33i60b34971.
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