What is it about?

There are links between the fetoplacental insufficiency, THs alterations, and immunosuppression. This state might be associated with the cardiovascular dysfunctions. This reduction results from the disruption of the HPTA, the energy expenditure and cytokine networks, and inducing oxidative stress, and cell death.

Featured Image

Why is it important?

Any disorders in the thyroid-adipokines axis might alter the fetoplacental communications. This can inhibit the passage of maternal nutrients to fetuses and delay the critical development.

Perspectives

Further additional studies are needed to detect the signaling of this axis during the gestation in human and animals.

Full Professor Ahmed R. G.
Division of Anatomy and Embryology, Zoology department, Faculty of Science, Beni-Suef University, Egypt.

Read the Original

This page is a summary of: Synergistic Actions of Thyroid-Adipokines Axis during Development, Endocrinology & Metabolic Syndrome, January 2017, OMICS Publishing Group,
DOI: 10.4172/2161-1017.1000280.
You can read the full text:

Read

Contributors

The following have contributed to this page