What is it about?
Increased activity of the renal sodium chloride cotransporter is linked to high blood pressure. The study of a rare genetic disease in which this cotransporter is more active has revealed new pathways that regulate it and change blood pressure. This review summarizes our current understanding of these pathways.
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This page is a summary of: Cullin 3/with No Lysine [K] Kinase/Ste20/SPS-Related Proline Alanine Rich Kinase Signaling: Impact on NaCl Cotransporter Activity in BP Regulation, Kidney360, August 2024, Wolters Kluwer Health,
DOI: 10.34067/kid.0000000000000527.
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