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We investigated S100B proteins, as they have been frequently reported elevated in autism patients. In this study, we could show that S100B acts as zinc binding protein which is activated by zinc binding to enable its protective role in the brain. However, in case of overload with S100B, the demand of S100B for zinc is high enough to lower zinc levels in the brain.
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This page is a summary of: Zinc Binding to S100B Affords Regulation of Trace Metal Homeostasis and Excitotoxicity in the Brain, Frontiers in Molecular Neuroscience, January 2018, Frontiers,
DOI: 10.3389/fnmol.2017.00456.
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