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We demonstrate that the long ncRNA DNM3os is elevated and acts as a promoter of fibrotic and inflammatory signalling in diabetic kidney disease, while microRNA-214 which is encoded by DNM3os acts as a negative regulator by targeting DIAPH1. These data suggest miR-214 may be of therapeutic value against diabetic kidney disease.

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This page is a summary of: miR-214 and its primary transcript Dnm3os regulate fibrosis and inflammation through RAGE signalling in diabetic kidney disease, Diabetes, April 2025, American Diabetes Association,
DOI: 10.2337/db24-0121.
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