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An increased rate of fatty acid beta oxidation in liver has been deemed as an essential event during the onset of obesity. We show that inhibited fatty acid oxidation in liver in fact triggers a protective response and send a signal to the adipose tissue to enhance energy expenditure.
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This page is a summary of: Hepatic CPT1A Facilitates Liver–Adipose Cross Talk via Induction of FGF21 in Mice, Diabetes, October 2021, American Diabetes Association,
DOI: 10.2337/db21-0363.
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