What is it about?
How hepatic mitochondria become dysfunctional during nonalcoholic fatty liver disease/steatohepatitis (NAFLD/NASH) development is unknown. We show that hepatocyte-specific endothelial nitric oxide synthase is a vital regulator of NAFLD/NASH susceptibility by maintaining a healthy mitochondrial pool within the liver with clinical relevance to patients.
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This page is a summary of: Critical Role for Hepatocyte-Specific eNOS in NAFLD and NASH, Diabetes, August 2021, American Diabetes Association,
DOI: 10.2337/db20-1228.
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