What is it about?

Increasing evidence supports the role of fat accumulation in the pancreas (i.e., pancreatic steatosis) in the development of type 2 diabetes, atherosclerosis, acute pancreatitis, and pancreatic cancer. In spite of this high medical relevance, and an estimated prevalence between 16 and 35%, the pathogenesis of non-alcoholic fatty pancreas disease (NAFPD) is still largely unknown. This study unravels a role for STK25 in determining the susceptibility to NAFPD in mice in connection to obesity.

Featured Image

Read the Original

This page is a summary of: Protein kinase STK25 aggravates the severity of non-alcoholic fatty pancreas disease in mice, Journal of Endocrinology, July 2017, Bioscientifica,
DOI: 10.1530/joe-17-0018.
You can read the full text:

Read

Contributors

Be the first to contribute to this page