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Neonatal oxygen exposure in rats is a model to study premature birth and related outcomes. Here we identify that neonatal oxygen exposure results in elevated pulmonary pressures (pulmonary hypertension) and late onset right ventricular dysfunction due to impaired mitochondrial function.
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This page is a summary of: Postnatal Hyperoxia Exposure Durably Impairs Right Ventricular Function and Mitochondrial Biogenesis, American Journal of Respiratory Cell and Molecular Biology, May 2017, American Thoracic Society,
DOI: 10.1165/rcmb.2016-0256oc.
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