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Cigarette smoke exposure triggers mitochondrial respiratory inhibition and oxidative stress. The use of an alternative respiratory enzyme relieves mitochondrial stress and attenuates lung dysfunction and tissue damage.

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This page is a summary of: Alternative Oxidase Attenuates Cigarette Smoke–induced Lung Dysfunction and Tissue Damage, American Journal of Respiratory Cell and Molecular Biology, May 2019, American Thoracic Society,
DOI: 10.1165/rcmb.2018-0261oc.
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