Biofield Treatment on Viral Load Against Human Immunodeficiency-1 and Cytomegalo Viruses

What is it about?

Viral load quantification is the amount of particular viral DNA or RNA in a blood samples. It is one of the surrogate biomarker of AIDS. High viral load indicates that the immune system is failed to fight against viruses. The aim of this study was to evaluate the impact of biofield treatment on HIV-1 and HCMV in terms of viral loads as surrogate marker. The viral load assay was performed on stored stock cultures of HIV infected human plasma samples before and after 7 days of biofield treatment using Roche COBAS® AMPLICOR analyzer. Viral load (HIV-1 RNA and HCMV DNAaemia) was considered as surrogate marker for assessment of the impact of Mr. Trivedi’s biofield treatment in HIV infected stored plasma samples. The viral load quantification of HIV-1 RNA in infected stored plasma samples was significantly reduced by 65% in biofield treated group as compared to control. Additionally, viral load of HCMV DNAaemia in infected stored plasma samples was also reduced by 80% in the biofield treated group as compared to control. Because, children are more prone to HCMV infection and adults are generally liable to suffer from HIV-1 infection. As the biofield treatment has reduced HCMV DNAaemia, it could be beneficial for HIV infected children populations. Altogether, data suggest that biofield treatment has significantly reduced the viral load quantification in HIV-1 and HCMV infected stored plasma samples and could be a suitable alternative treatment strategy for AIDS patients in near future.

Featured Image

Why is it important?

Human immunodeficiency virus type 1 (HIV-1) is the main causative agent of acquired immune deficiency syndrome (AIDS) [1]. HIV is a worldwide pandemic disease, and the number of infected peoples around the world increasing day by day. Recent estimate from World Health Organization (WHO) shows that 16.3 million people have died from AIDS since the beginning of the epidemic. Currently, around 34.3 million people alive with HIV infection, in which approximately 7% are young adults [2], infected with HIV type 1 (HIV-1) across the mucosal surfaces or by direct inoculation. The virus first attacks to dendritic cells (DCs) and subsequently spreads to cluster of differentiation - 4 (CD4+) T lymphocytes [3]. Human cytomegalovirus (HCMV) is a vernacular name of the human herpes virus - 5, a highly host-specific virus of the herpesviridae family rarely causes symptoms. The target DNA sequence is specific, located within the HCMV DNA polymerase gene, and is not homologous to other members of the human herpes virus family. The pregnant women and immune weakened persons are highly prone to acquire infection by HCMV virus. It spreads through various body fluids, such as blood, urine, saliva, semen, and breast milk. It may also causes serious morbidity and mortality in organ transplant recipients, immunocompromised, HIV infected patients and congenitally infected newborns [4]. HCMV is mainly prevalent in kidney transplant patients. The viral ‘threshold load’ is very sensitive and specific for predicting both white blood cell and platelet quantification in kidney transplant patients. The threshold value is more than 10000 copies/mL considered as HCMV infection [5]. In infected adults, HIV viral load is predictive of progression to AIDS [6, 7] and HCMV DNAaemia predicts progression to disease, particularly retinitis [8]. Several study reports have confirmed the relation between HIV-1 viral load and HCMV DNAaemia levels in terms of prediction of disease (AIDS) progression in both adults [6, 7] and children [9,10]. Researchers found an excellent correlation between two viruses i.e. HIV infected adults are highly predictable for the development of HCMV end organ disease [11]. Hence, in this experiment the dual viruses were taken into consideration as a rational. Immunomodulatory therapies are currently used against HIV-1 infections which include mycophenolate mofetil, cyclosporine, interleukin-2 (IL-2), and various vaccines [12]. Although several treatment strategie are available against AIDS patients infected by HIV-1 such as antiretroviral agents and vaccination. But some difficulties are also present. Based on above lacunas, there is a need of an alternative way which may be useful to determine the viral loads by either enhancing the application of existing agents or by means of some alternative strategy or developing new drugs. Biofield treatment is an alternative approach which may be useful to improve these unfilled space associated with AIDS infected patients. Mr. Trivedi’s unique biofield treatment (The Trivedi effect®) has been extensively contributes in scientific communities in several fields [13-16]. Therefore, authors interested, to evaluate the impact of biofield treatment on viral load in HIV and HCMV infected plasma samples.

Perspectives