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an aberrant remodeling of the pulmonary vasculature appears to have implications in IPF pathogenesis

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PF sera-induced cellular effects were significantly blunted in cells exposed to the NADPH oxidase inhibitor diphenyleneiodonium (DPI) proving the causative role of ROS and suggesting their potential cellular source. C

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This page is a summary of: Antioxidant Activity Mediates Pirfenidone Antifibrotic Effects in Human Pulmonary Vascular Smooth Muscle Cells Exposed to Sera of Idiopathic Pulmonary Fibrosis Patients, Oxidative Medicine and Cellular Longevity, October 2018, Hindawi Publishing Corporation,
DOI: 10.1155/2018/2639081.
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