What is it about?
Infective endocarditis is a biofilm-related infetious disease and refractory to antibiotic treatment. This study demonstrated the AtlA, autolysin of S. mutans, contributes to bacterial extracellular DNA release, which facilitate to biofilm formation on the damaged valve of experimental endocarditis rats.
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Why is it important?
Our finding demonstrated the role of bacterial AtlA and eDNA in the pathogenesis of IE. It provides important important information in the pathogenesis of IE.
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This page is a summary of: AtlA Mediates Extracellular DNA Release, Which Contributes to Streptococcus mutans Biofilm Formation in an Experimental Rat Model of Infective Endocarditis, Infection and Immunity, July 2017, ASM Journals,
DOI: 10.1128/iai.00252-17.
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