Deletion of collapsin response mediator protein 4 results in abnormal layer thickness and elongation of mitral cell apical dendrites in the neonatal olfactory bulb

What is it about?

Here we found that deletion of CRMP4, one of the CRMP family (CRMP1–5) responsible for Semaphorin signal transduction, result in altered dendritic elongation in mitral cells in the olfactory bulb (OB). This abnormality also changed layer thickness of the OB during early postnatal stage.

Featured Image