What is it about?
First: the mechanism of how elongated rete ridge is produced by continual neutrophil infiltration into the epidermis. Second: the mechanism of how IL-23/Th17 axis is generated by continual neutrophil infiltration into the epidermis. Third: anti-IL-17A and anti-IL23 antibodies exert therapeutic effects on psoriasis through blocking neutrophil mobilization from the bone marrow.
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This page is a summary of: Development of psoriasis by continual neutrophil infiltration into the epidermis, Experimental Dermatology, August 2018, Wiley,
DOI: 10.1111/exd.13746.
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