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The ultra-rapidly activating delayed rectifier K+ current IKur (Kv1.5) plays an important role in human atrial repolarization. We demonstrate that EGFR tyrosine kinase and soluble Src-family kinases have opposite regulation on human atrial IKur.
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This page is a summary of: Genistein and tyrphostin AG556 decrease ultra‐rapidly activating delayed rectifier K+ current of human atria by inhibiting EGF receptor tyrosine kinase, British Journal of Pharmacology, February 2017, Wiley,
DOI: 10.1111/bph.13710.
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