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  1. An intergenic non-coding RNA promoter required for histone modifications in the human β-globin chromatin domain
  2. Original Research: Generation of non-deletional hereditary persistence of fetal hemoglobin β-globin locus yeast artificial chromosome transgenic mouse models: -175 Black HPFH and -195 Brazilian HPFH
  3. O-GlcNAcase Expression is Sensitive to Changes in O-GlcNAc Homeostasis
  4. A Cell-Based High-Throughput Screen for Novel Chemical Inducers of Fetal Hemoglobin for Treatment of Hemoglobinopathies
  5. Extensively self-renewing erythroblasts derived from transgenic β-yac mice is a novel model system for studying globin switching and erythroid maturation
  6. Mi2β Is Required for γ-Globin Gene Silencing: Temporal Assembly of a GATA-1-FOG-1-Mi2 Repressor Complex in β-YAC Transgenic Mice
  7. Sickle Cell Disease: Genetics, Cellular and Molecular Mechanisms, and Therapies
  8. Induction of Fetal Hemoglobin In Vivo Mediated by a Synthetic γ-Globin Zinc Finger Activator
  9. Transgenic Expression of Human LAMA5 Suppresses Murine Lama5 mRNA and Laminin α5 Protein Deposition
  10. siDNMT1 increases γ-globin expression in chemical inducer of dimerization (CID)−dependent mouse βYAC bone marrow cells and in baboon erythroid progenitor cell cultures
  11. Mutation in erythroid specific transcription factor KLF1 causes Hereditary Spherocytosis in the Nan hemolytic anemia mouse model
  12. Positive selection of DNA-protein interactions in mammalian cells through phenotypic coupling with retrovirus production
  13. Preparation of intact yeast artificial chromosome DNA for transgenesis of mice
  14. Deletion of the human β-globin LCR 5′HS4 or 5′HS1 differentially affects β-like globin gene expression in β-YAC transgenic mice
  15. Human β-globin locus control region hypersensitive site specificity for globin gene activation during erythropoiesis
  16. Silencing of Aγ-globin gene expression during adult definitive erythropoiesis is mediated by GATA-1 binding
  17. Vertebrate Models for Sickle Cell Disease Research
  18. Identification of novel candidate genes for globin regulation in erythroid cells containing large deletions of the human β-globin gene cluster
  19. Transgenic mice carrying yeast artificial chromosomes
  20. Hemoglobin switching: new insights
  21. Locus control regions
  22. The 5′HS4 core element of the human β-globin locus control region is required for high-level globin gene expression in definitive but not in primitive erythropoiesis
  23. Single-Step Conversion of P1 and P1 Artificial Chromosome Clones into Yeast Artificial Chromosomes
  24. Locus control regions: coming of age at a decade plus
  25. [11] Use of yeast artificial chromosomes to express genes in transgenic mice
  26. Developmental Specificity of the Interaction between the Locus Control Region and Embryonic or Fetal Globin Genes in Transgenic Mice with an HS3 Core Deletion
  27. Production of transgenic mice with yeast artificial chromosomes
  28. Production and Analysis of Transgenic Mice Containing Yeast Artificial Chromosomes
  29. Generation of transgenic mice from yeast artificial chromosome DNA that has been modified by gene targeting
  30. Coexpression of ϵ, Gγ and Aγ Globin mRNA in Embryonic Red Blood Cells from a Single Copy β-YAC Transgenic Mouse
  31. Increased expression of the Escherichia coli umuDC operon restores SOS mutagenesis in lexA41 cells
  32. UV-INDUCIBLE SOS RESPONSE IN Escherichia coli
  33. Differential repression of SOS genes by unstable LexA41 (Tsl-1) protein causes a “split-phenotype” in Escherichia coli K-12
  34. Genetics of DNA repair in bacteria
  35. Differential Expression of SOS Genes in an E. Coli Mutant Producing Unstable LexA Protein Enhances Excision Repair But Inhibits Mutagenesis
  36. Viability of Escherichia coli K-12 DNA adenine methylase (dam) mutants requires increased expression of specific genes in the SOS regulon
  37. Production of Transgenic Mice with Yeast Artificial Chromosomes
  38. Establishment of Cell Lines That Exhibit Correct Ontogenic Stage-Specific Gene Expression Profiles From Tissues of Yeast Artificial Chromosome Transgenic Mice Using Chemically Induced Growth Signals