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  1. Inhibitory components of retinal bipolar cell receptive fields are differentially modulated by dopamine D1 receptors
  2. Reductions in Calcium Signaling Limit Inhibition to Diabetic Retinal Rod Bipolar Cells
  3. Dopamine D1 receptor activation reduces local inner retinal inhibition to light-adapted levels
  4. Dopamine D1 receptor activation contributes to light-adapted changes in retinal inhibition to rod bipolar cells
  5. Early Retinal Neuronal Dysfunction in Diabetic Mice: Reduced Light-Evoked Inhibition Increases Rod Pathway Signaling
  6. Light adaptation alters inner retinal inhibition to shape OFF retinal pathway signaling
  7. Different types of retinal inhibition have distinct neurotransmitter release properties
  8. Light adaptation alters the source of inhibition to the mouse retinal OFF pathway
  9. Slow changes in Ca2+ cause prolonged release from GABAergic retinal amacrine cells
  10. Inhibition to retinal rod bipolar cells is regulated by light levels
  11. Nonlinear Interactions between Excitatory and Inhibitory Retinal Synapses Control Visual Output
  12. Rod Vision Is Controlled by Dopamine-Dependent Sensitization of Rod Bipolar Cells by GABA
  13. Signal Processing in the Inner Retina
  14. Multiple pathways of inhibition shape bipolar cell responses in the retina
  15. Interneuron Circuits Tune Inhibition in Retinal Bipolar Cells
  16. Development of Presynaptic Inhibition Onto Retinal Bipolar Cell Axon Terminals Is Subclass-Specific
  17. Visual Signal Processing in the Inner Retina
  18. Presynaptic inhibition differentially shapes transmission in distinct circuits in the mouse retina
  19. Receptor and Transmitter Release Properties Set the Time Course of Retinal Inhibition
  20. GABAA, GABACand glycine receptor-mediated inhibition differentially affects light-evoked signalling from mouse retinal rod bipolar cells
  21. GABAC receptor-mediated inhibition in the retina
  22. Mechanisms for the Modulation of Native Glycine Receptor Channels by Ethanol
  23. Differential Effects of Ethanol on GABAA and Glycine Receptor-Mediated Synaptic Currents in Brain Stem Motoneurons