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  1. Modes of Human T Cell Leukemia Virus Type 1 Transmission, Replication and Persistence
  2. Mutation of a Single Envelope N-Linked Glycosylation Site Enhances the Pathogenicity of Bovine Leukemia Virus
  3. Plant polysaccharides initiate underground crosstalk with bacilli by inducing synthesis of the immunogenic lipopeptide surfactin
  4. The bovine leukemia virus microRNAs permit escape from innate immune response and contribute to viral replication in the natural host
  5. C-102 Vaccination against deltaretroviruses
  6. Abstract 3174: The host genomic environment of the provirus determines the abundance of BLV-infected clones
  7. Vaccination against δ-Retroviruses: The Bovine Leukemia Virus Paradigm
  8. Massive depletion of BLV proviral clones located in genomic transcriptionally active sites during primary infection
  9. Hyper-replicative bovine leukemia virus by mutation of an envelope N-linked glycosylation site
  10. Massive Depletion of Bovine Leukemia Virus Proviral Clones Located in Genomic Transcriptionally Active Sites during Primary Infection
  11. Chemoresistance to Valproate Treatment of Bovine Leukemia Virus-Infected Sheep; Identification of Improved HDAC Inhibitors
  12. Viral Expression Directs the Fate of B Cells in Bovine Leukemia Virus-Infected Sheep
  13. Preventive and Therapeutic Strategies for Bovine Leukemia Virus: Lessons for HTLV
  14. Role of glycosylation in bovine leukemia virus infection