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  1. Glycine-zipper motifs in hepatitis C virus nonstructural protein 4B are required for the establishment of viral replication organelles
  2. Hepatitis C Virus Replication Depends on Endosomal Cholesterol Homeostasis
  3. Cell-free expression, purification, and membrane reconstitution for NMR studies of the nonstructural protein 4B from hepatitis C virus
  4. Hepatitis C virus's next top models?
  5. Functional expression, purification, characterization, and membrane reconstitution of non-structural protein 2 from hepatitis C virus
  6. Flaviviridae Replication Organelles: Oh, What a Tangled Web We Weave
  7. NS5A Domain 1 and Polyprotein Cleavage Kinetics Are Critical for Induction of Double-Membrane Vesicles Associated with Hepatitis C Virus Replication
  8. The predominant species of nonstructural protein 4B in hepatitis C virus-replicating cells is not palmitoylated
  9. A sensor at the lipid‐protein interface: Lipid peroxidation controls hepatitis C virus replication
  10. Wheat germ cell-free expression: Two detergents with a low critical micelle concentration allow for production of soluble HCV membrane proteins
  11. Aminoterminal Amphipathic α-Helix AH1 of Hepatitis C Virus Nonstructural Protein 4B Possesses a Dual Role in RNA Replication and Virus Production
  12. Daclatasvir-Like Inhibitors of NS5A Block Early Biogenesis of Hepatitis C Virus–Induced Membranous Replication Factories, Independent of RNA Replication
  13. Hepatitis C Virus RNA Replication and Assembly: Living on the Fat of the Land
  14. Characterization of the Mode of Action of a Potent Dengue Virus Capsid Inhibitor
  15. Inhibition of HCV Replication by Cyclophilin Antagonists Is Linked to Replication Fitness and Occurs by Inhibition of Membranous Web Formation
  16. Purification of HCV-remodeled and Control ER Membranes
  17. Morphological and Biochemical Characterization of the Membranous Hepatitis C Virus Replication Compartment
  18. Architecture and biogenesis of plus-strand RNA virus replication factories
  19. NS4B Self-Interaction through Conserved C-Terminal Elements Is Required for the Establishment of Functional Hepatitis C Virus Replication Complexes