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  1. Lymph node stromal cells vary in susceptibility to infection but can support the intracellular growth of Listeria monocytogenes
  2. Egress of Listeria monocytogenes from Mesenteric Lymph Nodes Depends on Intracellular Replication and Cell-to-Cell Spread
  3. Enrichment of Neutrophils and Monocytes From the Liver Following Either Oral or Intravenous Listeria monocytogenes Infection
  4. Innate and Adaptive Immune Responses duringListeria monocytogenesInfection
  5. Neutrophils from Both Susceptible and Resistant Mice Efficiently Kill Opsonized Listeria monocytogenes
  6. Intracellular Listeria monocytogenes Comprises a Minimal but Vital Fraction of the Intestinal Burden following Foodborne Infection
  7. Cyclic di-GMP-dependent Signaling Pathways in the Pathogenic Firmicute Listeria monocytogenes
  8. Listeria monocytogenes: Cultivation and Laboratory Maintenance
  9. A Mouse Model of Foodborne Listeria monocytogenes Infection
  10. Fecal transplantation does not transfer either susceptibility or resistance to food borne listeriosis in C57BL/6 and BALB/c/By mice
  11. Oral Transmission of <em>Listeria monocytogenes</em> in Mice via Ingestion of Contaminated Food
  12. InlA Promotes Dissemination of Listeria monocytogenes to the Mesenteric Lymph Nodes during Food Borne Infection of Mice
  13. Lymphocytes serve as a reservoir for Listeria monocytogenes growth during infection of mice
  14. Multiple Mechanisms Contribute to the Robust Rapid Gamma Interferon Response by CD8+ T Cells during Listeria monocytogenes Infection
  15. Use of the CD107 mobilization assay reveals that cytotoxic T lymphocytes with novel MHC-Ib restriction are activated during Listeria monocytogenes infection
  16. H2-M3–restricted CD8+ T cells are not required for MHC class Ib–restricted immunity against Listeria monocytogenes
  17. TheListeria monocytogenes lemA Gene Product Is Not Required forIntracellular Infection or To Activate fMIGWII-Specific TCells
  18. Multigenic control of Listeria monocytogenes susceptibility in mice