All Stories

  1. Fibroblasts from recurrent fibrotic overgrowths reveal high rate of proliferation in vitro - findings from the study of hereditary and idiopathic gingival fibromatosis
  2. TIMP-1 association with collagen type I overproduction in hereditary gingival fibromatosis
  3. Association of Distinct Fine Specificities of Anti−Citrullinated Peptide Antibodies With Elevated Immune Responses to Prevotella intermedia in a Subgroup of Patients With Rheumatoid Arthritis and Periodontitis
  4. Analysis of mutations in the SOS-1 gene in two Polish families with hereditary gingival fibromatosis
  5. Peptidylarginine deiminase fromPorphyromonas gingivaliscontributes to infection of gingival fibroblasts and induction of prostaglandin E2-signaling pathway
  6. Porphyromonas gingivalis Facilitates the Development and Progression of Destructive Arthritis through Its Unique Bacterial Peptidylarginine Deiminase (PAD)
  7. Use of Sensitive, Broad-Spectrum Molecular Assays and Human Airway Epithelium Cultures for Detection of Respiratory Pathogens
  8. Persistence of intracellular and extracellular changes after incompletely suppressing expression of the R789C (p.R989C) and R992C (p.R1192C) collagen II mutants
  9. Reducing the effects of intracellular accumulation of thermolabile collagen II mutants by increasing their thermostability in cell culture conditions
  10. R992C (p.R1192C) Substitution in Collagen II Alters the Structure of Mutant Molecules and Induces the Unfolded Protein Response
  11. Collagen II R992C mutation leads to unfolded protein response
  12. Cellular toxicity of oxycholesterols
  13. Targeting of solid tumors and blood malignancies by antibody-based therapies — EGFR-pathway as an example