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  1. Transcriptional co-repressor SIN3A silencing rescues decline in memory consolidation during scopolamine-induced amnesia
  2. Epigenetic Regulation of Memory-Therapeutic Potential for Disorders
  3. Differential proteome profiling in the hippocampus of amnesic mice
  4. Presenilin 2 overexpression is associated with apoptosis in Neuro2a cells
  5. Hippocampal chromatin-modifying enzymes are pivotal for scopolamine-induced synaptic plasticity gene expression changes and memory impairment
  6. Epigenetic regulation of presenilin 1 and 2 in the cerebral cortex of mice during development