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  1. Endocannabinoid system in neurodegenerative disorders
  2. Postnatal ethanol exposure alters levels of 2-arachidonylglycerol-metabolizing enzymes and pharmacological inhibition of monoacylglycerol lipase does not cause neurodegeneration in neonatal mice
  3. CB1-receptor knockout neonatal mice are protected against ethanol-induced impairments of DNMT1, DNMT3A, and DNA methylation
  4. Major Enzymes of Endocannabinoid Metabolism
  5. Elevation of endogenous anandamide impairs LTP, learning, and memory through CB1 receptor signaling in mice
  6. CB1 receptor-mediated signaling underlies the hippocampal synaptic, learning, and memory deficits following treatment with JWH-081, a new component of spice/K2 preparations