All Stories

  1. Sodium channels
  2. From plant extract to molecular panacea: a commentary on Stone (1763) 'An account of the success of the bark of the willow in the cure of the agues'
  3. Pain without Nociceptors? Nav1.7-Independent Pain Mechanisms
  4. Novel Mutations Mapping to the Fourth Sodium Channel Domain of Nav1.7 Result in Variable Clinical Manifestations of Primary Erythromelalgia
  5. Splice Variants of NaV1.7 Sodium Channels Have Distinct β Subunit-Dependent Biophysical Properties
  6. Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons
  7. Loss-of-function mutations in sodium channel Nav1.7 cause anosmia
  8. Nociceptor-specific gene deletion reveals a major role for Na v 1.7 (PN1) in acute and inflammatory pain