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  1. The MEC1 and MEC2 Lines Represent Two CLL Subclones in Different Stages of Progression towards Prolymphocytic Leukemia
  2. T Cells Modulate Epstein-Barr Virus Latency Phenotypes during Infection of Humanized Mice
  3. Cytomegalovirus-Seropositive Children Show Inhibition of In Vitro EBV Infection That Is Associated with CD8+CD57+ T Cell Enrichment and IFN- 
  4. The role of DNA hypomethylation, histone acetylation and in vivo protein-DNA binding in Epstein–Barr virus-induced CD23 upregulation
  5. EBV genome carrying B lymphocytes that express the nuclear protein EBNA-2 but not LMP-1
  6. p53 contributes to T cell homeostasis through the induction of pro-apoptotic SAP
  7. Simultaneous detection of the two main proliferation driving EBV encoded proteins, EBNA-2 and LMP-1 in single B cells
  8. Type I interferons directly down-regulate BCL-6 in primary and transformed germinal center B cells: Differential regulation in B cell lines derived from endemic or sporadic Burkitt’s lymphoma
  9. Soluble factors produced by activated CD4+T cells modulate EBV latency
  10. Monocytes enhance cell proliferation and LMP1 expression of nasal natural killer/T-cell lymphoma cells by cell contact-dependent interaction through membrane-bound IL-15
  11. Restricted expression of EBV encoded proteins in in vitro infected CLL cells
  12. Chronic lymphoid leukemia cells are highly sensitive to the combination of prednisolone and daunorubicin, but much less to doxorubicin or epirubicin
  13. Exosomes Containing Glycoprotein 350 Released by EBV-Transformed B Cells Selectively Target B Cells through CD21 and Block EBV Infection In Vitro
  14. STAT6 signaling pathway activated by the cytokines IL-4 and IL-13 induces expression of the Epstein-Barr virus-encoded protein LMP-1 in absence of EBNA-2: implications for the type II EBV latent gene expression in Hodgkin lymphoma
  15. Deficiency of the proapoptotic SAP function in X-linked lymphoproliferative disease aggravates Epstein-Barr virus (EBV) induced mononucleosis and promotes lymphoma development
  16. IL-21 imposes a type II EBV gene expression on type III and type I B cells by the repression of C- and activation of LMP-1-promoter
  17. To the genesis of Burkitt lymphoma: Regulation of apoptosis by EBNA-1 and SAP may determine the fate of Ig-myc translocation carrying B lymphocytes
  18. The apoptosis modulating role of SAP (SLAM associated protein) contributes to the symptomatology of the X linked lymphoproliferative disease
  19. Cytokine mediated induction of the major Epstein–Barr virus (EBV)-encoded transforming protein, LMP-1
  20. Concomitant increase of LMP1 and CD25 (IL-2-receptor α) expression induced by IL-10 in the EBV-positive NK lines SNK6 and KAI3
  21. EBV Infection Induces Expression of the Transcription Factors ATF-2/c-Jun in B Lymphocytes but not in B-CLL Cells
  22. Upregulation of LMP1 Expression by Histone Deacetylase Inhibitors in an EBV Carrying NPC Cell Line
  23. In vitro EBV-infected subline of KMH2, derived from Hodgkin lymphoma, expresses only EBNA-1, while CD40 ligand and IL-4 induce LMP-1 but not EBNA-2
  24. SH2D1A expression in Burkitt lymphoma cells is restricted to EBV positive group I lines and is downregulated in parallel with immunoblastic transformation
  25. The X-linked lymphoproliferative disease gene product SAP is expressed in activated T and NK cells
  26. Intranuclear Localization of the Transcription Coadaptor CBP/p300 and the Transcription Factor RBP-Jk in Relation to EBNA-2 and -5 in B Lymphocytes
  27. EBNA promoter usage in ebv-negative burkitt lymphoma cell lines converted with a neomycin-resistant EBV strain
  28. SH2D1A and slam protein expression in human lymphocytes and derived cell lines
  29. SH2D1A and slam protein expression in human lymphocytes and derived cell lines
  30. LMP-1, the Epstein–Barr virus-encoded oncogene with a B cell activating mechanism similar to CD40