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  1. TDRD3 is an antiviral restriction factor that promotes IFN signaling with G3BP1
  2. Casein Kinase 2 Is Linked to Stress Granule Dynamics through Phosphorylation of the Stress Granule Nucleating Protein G3BP1
  3. Stress Granules Regulate Double-Stranded RNA-Dependent Protein Kinase Activation through a Complex Containing G3BP1 and Caprin1
  4. The Stress Granule Protein G3BP1 Recruits Protein Kinase R To Promote Multiple Innate Immune Antiviral Responses
  5. Cytoplasmic RNA Granules and Viral Infection
  6. Poliovirus-Mediated Shutoff of Host Translation: an Indirect Effect
  7. mRNA Decapping Enzyme 1a (Dcp1a)-induced Translational Arrest through Protein Kinase R (PKR) Activation Requires the N-terminal Enabled Vasodilator-stimulated Protein Homology 1 (EVH1) Domain
  8. Regulation of stress granules and P-bodies during RNA virus infection
  9. Diversion of stress granules and P-bodies during viral infection
  10. Large G3BP-induced granules trigger eIF2  phosphorylation
  11. How Do Viruses Interact with Stress-Associated RNA Granules?
  12. Regulation of stress granules in virus systems
  13. Animal virus schemes for translation dominance
  14. Poliovirus Unlinks TIA1 Aggregation and mRNA Stress Granule Formation
  15. Poliovirus Switches to an eIF2-Independent Mode of Translation during Infection
  16. Poliovirus-Mediated Disruption of Cytoplasmic Processing Bodies
  17. Cleavage of eukaryotic initiation factor eIF5B by enterovirus 3C proteases
  18. Cleavage of Poly(A)-Binding Protein by Poliovirus 3C Proteinase Inhibits Viral Internal Ribosome Entry Site-Mediated Translation
  19. Modulation of enteroviral proteinase cleavage of poly(A)-binding protein (PABP) by conformation and PABP-associated factors
  20. Translation of cIAP2 mRNA Is Mediated Exclusively by a Stress-Modulated Ribosome Shunt
  21. Inhibition of Cytoplasmic mRNA Stress Granule Formation by a Viral Proteinase
  22. Translational control by viral proteinases
  23. Translation of Eukaryotic Translation Initiation Factor 4GI (eIF4GI) Proceeds from Multiple mRNAs Containing a Novel Cap-dependent Internal Ribosome Entry Site (IRES) That Is Active during Poliovirus Infection
  24. Calicivirus 3C-Like Proteinase Inhibits Cellular Translation by Cleavage of Poly(A)-Binding Protein
  25. Proteolytic Cleavage of the Catalytic Subunit of DNA-Dependent Protein Kinase during Poliovirus Infection
  26. BCL-2 Translation Is Mediated via Internal Ribosome Entry during Cell Stress
  27. Translation of cellular inhibitor of apoptosis protein 1 (c-IAP1) mRNA is IRES mediated and regulated during cell stress
  28. Cleavage of Poly(A)-Binding Protein by Poliovirus 3C Protease Inhibits Host Cell Translation: a Novel Mechanism for Host Translation Shutoff
  29. Generation of Multiple Isoforms of Eukaryotic Translation Initiation Factor 4GI by Use of Alternate Translation Initiation Codons
  30. Efficient Cleavage of Ribosome-Associated Poly(A)-Binding Protein by Enterovirus 3C Protease
  31. Multiple eIF4GI-Specific Protease Activities Present in Uninfected and Poliovirus-Infected Cells
  32. The Predominant eIF4G-Specific Cleavage Activity in Poliovirus-Infected HeLa Cells Is Distinct from 2A Protease
  33. Direct Cleavage of eIF4G by Poliovirus 2A Protease Is Inefficientin Vitro
  34. K562 Cell Strains Differ in Their Response to Poliovirus Infection
  35. Persistent Infection of Human Erythroblastoid Cells by Poliovirus
  36. Characterization of the roles of conserved cysteine and histidine residues in poliovirus 2A protease
  37. Identification of essential amino acid residues in the functional activity of poliovirus 2A protease
  38. Cleavage of the cap binding protein complex polypeptide p220 is not effected by the second poliovirus protease 2A
  39. Poliovirus protease does not mediate cleavage of the 220,000-Da component of the cap binding protein complex.