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  1. O-GlcNAcylation Signal Mediates Proteasome Inhibitor Resistance in Cancer Cells by Stabilizing NRF1
  2. Effects of deficiency of Kelch-like ECH-associated protein 1 on skeletal organization: a mechanism for diminished nuclear factor of activated T cells cytoplasmic 1 during osteoclastogenesis
  3. Derepression of the DNA Methylation Machinery of the Gata1 Gene Triggers the Differentiation Cue for Erythropoiesis
  4. A Homeostatic Shift Facilitates Endoplasmic Reticulum Proteostasis through Transcriptional Integration of Proteostatic Stress Response Pathways
  5. Renal Anemia Model Mouse Established by Transgenic Rescue with an Erythropoietin Gene Lacking Kidney-Specific Regulatory Elements
  6. Absolute Amounts and Status of the Nrf2-Keap1-Cul3 Complex within Cells
  7. Nrf2-Mediated Regulation of Skeletal Muscle Glycogen Metabolism
  8. The Mediator Subunit MED16 Transduces NRF2-Activating Signals into Antioxidant Gene Expression
  9. Characterizations of Three Major Cysteine Sensors of Keap1 in Stress Response
  10. Whole-BodyIn VivoMonitoring of Inflammatory Diseases Exploiting Human Interleukin 6-Luciferase Transgenic Mice
  11. The HumanGATA1Gene Retains a 5′ Insulator That Maintains Chromosomal Architecture andGATA1Expression Levels in Splenic Erythroblasts
  12. Progenitor Stage-Specific Activity of acis-Acting Double GATA Motif forGata1Gene Expression
  13. Transcription Factor Nrf1 Negatively Regulates the Cystine/Glutamate Transporter and Lipid-Metabolizing Enzymes
  14. GATA2 Regulates Body Water Homeostasis through Maintaining Aquaporin 2 Expression in Renal Collecting Ducts
  15. Nrf2 Enhances Cholangiocyte Expansion in Pten-Deficient Livers
  16. Kinetic, Thermodynamic, and Structural Characterizations of the Association between Nrf2-DLGex Degron and Keap1
  17. The Keap1-Nrf2 System Prevents Onset of Diabetes Mellitus
  18. NF-E2 p45 Is Important for Establishing Normal Function of Platelets
  19. Regulatory Nexus of Synthesis and Degradation Deciphers Cellular Nrf2 Expression Levels
  20. Disruption of the Hbs1l-Myb Locus Causes Hereditary Persistence of Fetal Hemoglobin in a Mouse Model