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  1. Analyzing aggregation propensities of clinically relevant PTEN mutants: a new culprit in pathogenesis of cancer and other PTENopathies
  2. PTEN Physically Interacts with and Regulates E2F1-mediated Transcription in Lung Cancer
  3. Presence and utility of intrinsically disordered regions in kinases
  4. The PTEN Long N-tail is intrinsically disordered: increased viability for PTEN therapy