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  1. Targeting of Extracellular RNA Reduces Edema Formation and Infarct Size and Improves Survival After Myocardial Infarction in Mice
  2. cAMP controls the restoration of endothelial barrier function after thrombin-induced hyperpermeability via Rac1 activation
  3. P192Opposing effects of cAMP/PKA and cAMP/Epac signalling on in vitro angiogenesis: role of Rho gtpases
  4. P181cAMP-mediated Rac1 activation regulates the re-establishment of endothelial adherens junctions and barrier restoration during inflammation
  5. Impact of caloric restriction on myocardial ischaemia/reperfusion injury and new therapeutic options to mimic its effects
  6. Nucleoside triphosphates inhibit ADP, collagen, and epinephrine-induced platelet aggregation: Role of P2Y1 and P2Y12 receptors
  7. Internalization of Bacillus intermedius ribonuclease (BINASE) induces human alveolar adenocarcinoma cell death
  8. Hypoxia–reoxygenation‐induced endothelial barrier failure: role of RhoA, Rac1 and myosin light chain kinase
  9. Extracellular ATP attenuates ischemia-induced caspase-3 cleavage in human endothelial cells
  10. Opposing effects of ATP and adenosine on barrier function of rat coronary microvasculature
  11. Intermedin (adrenomedullin2) stabilizes the endothelial barrier and antagonizes thrombin‐induced barrier failure in endothelial cell monolayers
  12. Intermedin induces loss of coronary microvascular endothelial barrier via derangement of actin cytoskeleton: role of RhoA and Rac1
  13. The Role Of Neutrophil Extracellular Trap (NET) In Lung Epithelial Cell Cytotoxicity And Permeability
  14. cAMP/PKA antagonizes thrombin-induced inactivation of endothelial myosin light chain phosphatase: role of CPI-17
  15. Intermedin/adrenomedullin-2 is a hypoxia-induced endothelial peptide that stabilizes pulmonary microvascular permeability
  16. Extracellular ATP induces assembly and activation of the myosin light chain phosphatase complex in endothelial cells☆
  17. Accumulation of extracellular ATP protects against acute reperfusion injury in rat heart endothelial cells☆