All Stories

  1. CaMKII controls neuromodulation via neuropeptide gene expression and axonal targeting of neuropeptide vesicles
  2. CAPS-1 promotes fusion competence of stationary dense-core vesicles in presynaptic terminals of mammalian neurons
  3. Munc18-1 is a dynamically regulated PKC target during short-term enhancement of transmitter release
  4. A closer look at FBXO41 as a Parkinson's disease risk factor
  5. Unfolded protein response activates glycogen synthase kinase-3 via selective lysosomal degradation
  6. Synapse Associated Protein 102 (SAP102) Binds the C-Terminal Part of the Scaffolding Protein Neurobeachin
  7. Munc18-1 mutations that strongly impair SNARE-complex binding support normal synaptic transmission
  8. Crashpilot Underachieves due to Acetylation at the Nerve Terminal
  9. Deletion of Munc18-1 in 5-HT Neurons Results in Rapid Degeneration of the 5-HT System and Early Postnatal Lethality
  10. Corrigendum to “Automated analysis of neuronal morphology, synapse number and synaptic recruitment” [J. Neurosci. Methods 195 (2) (2011) 185–193]
  11. Automated analysis of neuronal morphology, synapse number and synaptic recruitment
  12. Munc18 and Munc13 regulate early neurite outgrowth
  13. Automated quantification of cellular traffic in living cells
  14. Munc18-1 in secretion: lonely Munc joins SNARE team and takes control
  15. Regulated exocytosis: merging ideas on fusing membranes
  16. The Role of Rab3a in Secretory Vesicle Docking Requires Association/Dissociation of Guanidine Phosphates and Munc18-1
  17. Interdependence of PKC-Dependent and PKC-Independent Pathways for Presynaptic Plasticity
  18. Docking of Secretory Vesicles Is Syntaxin Dependent
  19. Dissecting docking and tethering of secretory vesicles at the target membrane
  20. Vesicular Trafficking of Semaphorin 3A is Activity‐Dependent and Differs Between Axons and Dendrites
  21. Munc18–1 stabilizes syntaxin 1, but is not essential for syntaxin 1 targeting and SNARE complex formation
  22. Trophic support delays but does not prevent cell‐intrinsic degeneration of neurons deficient for munc18‐1
  23. Vesicle trafficking: pleasure and pain from SM genes
  24. Munc18-1 Promotes Large Dense-Core Vesicle Docking