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  1. Translating neuronal activity at the synapse: presynaptic calcium sensors in short-term plasticity
  2. Protein kinase C is a calcium sensor for presynaptic short-term plasticity
  3. Calcium-Dependent PKC Isoforms Have Specialized Roles in Short-Term Synaptic Plasticity
  4. Promoter Decommissioning by the NuRD Chromatin Remodeling Complex Triggers Synaptic Connectivity in the Mammalian Brain
  5. Calcium-Dependent PKC Isoforms Have Specialized Roles in Short-Term Synaptic Plasticity
  6. Cellular and molecular mechanisms of associative and nonassociative learning
  7. Calcium-Dependent Isoforms of Protein Kinase C Mediate Glycine-Induced Synaptic Enhancement at the Calyx of Held
  8. Adaptive Regulation Maintains Posttetanic Potentiation at Cerebellar Granule Cell Synapses in the Absence of Calcium-Dependent PKC
  9. Calcium-Dependent Isoforms of Protein Kinase C Mediate Posttetanic Potentiation at the Calyx of Held
  10. Protein degradation and memory formation
  11. Short-term forms of presynaptic plasticity
  12. Dynamics of Fast and Slow Inhibition from Cerebellar Golgi Cells Allow Flexible Control of Synaptic Integration
  13. Post‐translational regulation of an Aplysia glutamate transporter during long‐term facilitation
  14. The Ubiquitin-Proteasome System Is Necessary for Long-Term Synaptic Depression in Aplysia
  15. Synapsin Regulates Basal Synaptic Strength, Synaptic Depression, and Serotonin-Induced Facilitation of Sensorimotor Synapses in Aplysia
  16. Differential Expression of Posttetanic Potentiation and Retrograde Signaling Mediate Target-Dependent Short-Term Synaptic Plasticity
  17. The 5-HT- and FMRFa-activated signaling pathways interact at the level of the Erk MAPK cascade: Potential inhibitory constraints on memory formation
  18. cAMP-response Elements in Aplysia creb1, creb2, and Ap-uch Promoters: IMPLICATIONS FOR FEEDBACK LOOPS MODULATING LONG TERM MEMORY
  19. Cellular and molecular mechanisms of associative and nonassociative learning