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  1. Non-voltage-gated Ca2+ entry pathways in the heart: the untold STOrai?
  2. Modification of STIM1 by O-linked N-Acetylglucosamine (O-GlcNAc) Attenuates Store-operated Calcium Entry in Neonatal Cardiomyocytes
  3. Chronic ingestion of a Western diet increases O-linked-β-N-acetylglucosamine (O-GlcNAc) protein modification in the rat heart
  4. O-GlcNAc Modification of NFκB p65 Inhibits TNF-α-Induced Inflammatory Mediator Expression in Rat Aortic Smooth Muscle Cells
  5. Inhibition of O-GlcNAcase in perfused rat hearts by NAG-thiazolines at the time of reperfusion is cardioprotective in an O-GlcNAc-dependent manner
  6. Glycopeptide-specific monoclonal antibodies suggest new roles for O-GlcNAc
  7. Increased protein O-GlcNAc modification inhibits inflammatory and neointimal responses to acute endoluminal arterial injury
  8. Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein O-GlcNAc and increased mitochondrial Bcl-2
  9. Increased O-GlcNAc levels during reperfusion lead to improved functional recovery and reduced calpain proteolysis
  10. THE PROTECTIVE EFFECTS OF PUGNAC ON CARDIAC FUNCTION AFTER TRAUMA-HEMORRHAGE ARE MEDIATED VIA INCREASED PROTEIN O-GlcNAc LEVELS
  11. Glutamine-induced protection of isolated rat heart from ischemia/reperfusion injury is mediated via the hexosamine biosynthesis pathway and increased protein O-GlcNAc levels
  12. Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associated O-GlcNAc
  13. Increased hexosamine biosynthesis and protein O-GlcNAc levels associated with myocardial protection against calcium paradox and ischemia