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  1. The key role of Akt protein kinase in metabolic-inflammatory pathways cross-talk: TNF-α down-regulation and improving of insulin resistance in HepG2 cell line
  2. Possible improvement of neuronal mitochondrial bioenergetics by nobiletin involves activation of matrix substrate-level phosphorylation
  3. Nobiletin restores impaired hippocampal mitochondrial bioenergetics in hypothyroidism through activation of matrix substrate-level phosphorylation
  4. Thyroid hormones differentially regulate phosphorylation of ERK and Akt via integrinαvβ3 receptor in undifferentiated and differentiated PC-12 cells
  5. Adhesive Properties and Inflammatory Potential of Citrullinated Myelin Basic Protein Peptide 45–89
  6. Sodium nitroprusside, a nitric oxide donor, fails to bypass the block of neuronal differentiation in PC12 cells imposed by a dominant negative Ras protein
  7. NOBILETIN TRANSIENTLY INCREASES THE PRODUCTION OF NITRIC OXIDE AND CHANGES THE ACTIVITY OF SUCCINATE DEHYDROGENASE IN HUMAN BLOOD LYMPHOCYTES
  8. Social isolation in rats inhibits oxidative metabolism, decreases the content of mitochondrial K-Ras and activates mitochondrial hexokinase
  9. N-Methyl-<i>D</i>-Aspartate and σ-Ligands Change the Production of Interleukins 8 and 10 in Lymphocytes through Modulation of the NMDA Glutamate Receptor
  10. WITHDRAWN: Role of nitrosylaton and farnesylation in the regulation of homocysteine metabolism in PC12 cells
  11. l-NAME has Opposite Effects on the Productions of S-adenosylhomocysteine and S-adenosylmethionine in V12-H-Ras and M-CR3B-Ras Pheochromocytoma Cells
  12. Creatine enhances survival of glutamate-treated neuronal/glial cells, modulates Ras/NF-?B signaling, and increases the generation of reactive oxygen species
  13. ON EFFECTS OF Ca-BINDING PROTEINS AND BIOGENIC AMINES
  14. THE STUDY OF SYNTHESIS OF ACETYLPHOSPHOCHOLINE IN THE BRAIN