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  1. Candidalysin Is Required for Neutrophil Recruitment and Virulence During Systemic Candida albicans Infection
  2. EphA2 Is a Neutrophil Receptor for Candida albicans that Stimulates Antifungal Activity during Oropharyngeal Infection
  3. Selection of Candida albicans trisomy during oropharyngeal infection results in a commensal-like phenotype
  4. Candida albicans at Host Barrier Sites: Pattern Recognition Receptors and Beyond
  5. Selection of Candida albicans Trisomy during Oropharyngeal Infection Results in a Commensal-Like Phenotype
  6. Inhibition of EGFR Signaling Protects from Mucormycosis
  7. Author Correction: EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  8. The Hyr1 protein from the fungus Candida albicans is a cross kingdom immunotherapeutic target for Acinetobacter bacterial infection
  9. Candida albicans White-Opaque Switching Influences Virulence but Not Mating during Oropharyngeal Candidiasis
  10. Publisher Correction: EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  11. EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  12. Innate Immunity to Mucosal Candida Infections
  13. The Aryl Hydrocarbon Receptor Governs Epithelial Cell Invasion during Oropharyngeal Candidiasis
  14. Oropharyngeal Candidiasis: Fungal Invasion and Epithelial Cell Responses
  15. AMPlified Defense: Antimicrobial Peptides During Candida albicans Infection
  16. Aspergillus fumigatus CalA binds to integrin α5β1 and mediates host cell invasion
  17. Candida albicansresponds to glycostructure damage by Ace2-mediated feedback regulation of Cek1 signaling
  18. Domains of CaMsb2
  19. Candida and AMPs
  20. C. albicans Mucin Msb2
  21. Msb2 Shedding